"Repressor Proteins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Proteins which maintain the transcriptional quiescence of specific GENES or OPERONS. Classical repressor proteins are DNA-binding proteins that are normally bound to the OPERATOR REGION of an operon, or the ENHANCER SEQUENCES of a gene until a signal occurs that causes their release.
| Descriptor ID |
D012097
|
| MeSH Number(s) |
D12.776.260.703 D12.776.930.780
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Repressor Proteins".
Below are MeSH descriptors whose meaning is more specific than "Repressor Proteins".
This graph shows the total number of publications written about "Repressor Proteins" by people in this website by year, and whether "Repressor Proteins" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1995 | 5 | 4 | 9 |
| 1996 | 3 | 1 | 4 |
| 1997 | 5 | 0 | 5 |
| 1998 | 6 | 4 | 10 |
| 1999 | 11 | 4 | 15 |
| 2000 | 7 | 8 | 15 |
| 2001 | 11 | 9 | 20 |
| 2002 | 14 | 8 | 22 |
| 2003 | 15 | 8 | 23 |
| 2004 | 12 | 10 | 22 |
| 2005 | 16 | 8 | 24 |
| 2006 | 10 | 6 | 16 |
| 2007 | 15 | 6 | 21 |
| 2008 | 11 | 14 | 25 |
| 2009 | 11 | 10 | 21 |
| 2010 | 9 | 10 | 19 |
| 2011 | 15 | 5 | 20 |
| 2012 | 9 | 13 | 22 |
| 2013 | 14 | 9 | 23 |
| 2014 | 17 | 11 | 28 |
| 2015 | 13 | 15 | 28 |
| 2016 | 13 | 6 | 19 |
| 2017 | 13 | 7 | 20 |
| 2018 | 13 | 10 | 23 |
| 2019 | 14 | 4 | 18 |
| 2020 | 7 | 9 | 16 |
| 2021 | 11 | 6 | 17 |
| 2022 | 4 | 13 | 17 |
| 2023 | 0 | 5 | 5 |
| 2024 | 7 | 5 | 12 |
| 2025 | 2 | 3 | 5 |
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Below are the most recent publications written about "Repressor Proteins" by people in Profiles.
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Muscle Rev-erb controls time-dependent adaptations to chronic exercise in mice. Nat Commun. 2025 Jul 01; 16(1):5708.
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Alternative splicing of the Snap23 microexon is regulated by MBNL, QKI, and RBFOX2 in a tissue-specific manner and is altered in striated muscle diseases. RNA Biol. 2025 Dec; 22(1):1-20.
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Clonal hematopoiesis of indeterminate potential and incidence of venous thromboembolism in older adults. J Thromb Haemost. 2025 Jul; 23(7):2235-2241.
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De novo variants in RYBP are associated with a severe neurodevelopmental disorder and congenital anomalies. Genet Med. 2025 Apr; 27(4):101369.
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Co-Occurrence of ETV6::RUNX1 and P2RY8::CRLF2 Fusion in a Patient with Relapsed Acute B Lymphoblastic Leukemia. Ann Clin Lab Sci. 2025 Jan; 55(1):133-141.
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Clonal hematopoiesis-related mutant ASXL1 promotes atherosclerosis in mice via dysregulated innate immunity. Nat Cardiovasc Res. 2024 Dec; 3(12):1568-1583.
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Nerve injury inhibits Oprd1 and Cnr1 transcription through REST in primary sensory neurons. Sci Rep. 2024 11 04; 14(1):26612.
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ASXL1/TET2 genotype-based risk stratification outperforms ASXL1 mutational impact and is independent of mutant variant allele fractions in chronic myelomonocytic leukemia. Haematologica. 2024 Oct 01; 109(10):3419-3425.
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Epigenomic and Transcriptomic Profiling of Solitary Fibrous Tumors Identifies Site-Specific Patterns and Candidate Genes Regulated by DNA Methylation. Lab Invest. 2024 11; 104(11):102146.
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Trichorhinophalangeal syndrome 1 expression in breast and nonbreast metastases from M?llerian, lung, gastrointestinal tract, and pancreatic primary tumors by immunohistochemistry with cytology cell block specimens. Cancer Cytopathol. 2024 Dec; 132(12):799-808.