"Repressor Proteins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Proteins which maintain the transcriptional quiescence of specific GENES or OPERONS. Classical repressor proteins are DNA-binding proteins that are normally bound to the OPERATOR REGION of an operon, or the ENHANCER SEQUENCES of a gene until a signal occurs that causes their release.
| Descriptor ID |
D012097
|
| MeSH Number(s) |
D12.776.260.703 D12.776.930.780
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Repressor Proteins".
Below are MeSH descriptors whose meaning is more specific than "Repressor Proteins".
This graph shows the total number of publications written about "Repressor Proteins" by people in this website by year, and whether "Repressor Proteins" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 4 | 3 | 7 |
| 1995 | 12 | 9 | 21 |
| 1996 | 8 | 2 | 10 |
| 1997 | 18 | 2 | 20 |
| 1998 | 12 | 6 | 18 |
| 1999 | 22 | 7 | 29 |
| 2000 | 19 | 12 | 31 |
| 2001 | 22 | 11 | 33 |
| 2002 | 27 | 14 | 41 |
| 2003 | 30 | 16 | 46 |
| 2004 | 27 | 19 | 46 |
| 2005 | 34 | 16 | 50 |
| 2006 | 28 | 12 | 40 |
| 2007 | 26 | 14 | 40 |
| 2008 | 20 | 21 | 41 |
| 2009 | 20 | 22 | 42 |
| 2010 | 16 | 19 | 35 |
| 2011 | 35 | 12 | 47 |
| 2012 | 22 | 21 | 43 |
| 2013 | 20 | 18 | 38 |
| 2014 | 25 | 13 | 38 |
| 2015 | 21 | 16 | 37 |
| 2016 | 24 | 11 | 35 |
| 2017 | 20 | 11 | 31 |
| 2018 | 22 | 11 | 33 |
| 2019 | 19 | 6 | 25 |
| 2020 | 11 | 12 | 23 |
| 2021 | 21 | 7 | 28 |
| 2022 | 4 | 22 | 26 |
| 2023 | 1 | 18 | 19 |
| 2024 | 5 | 9 | 14 |
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Below are the most recent publications written about "Repressor Proteins" by people in Profiles.
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ASXL1/TET2 genotype-based risk stratification outperforms ASXL1 mutational impact and is independent of mutant variant allele fractions in chronic myelomonocytic leukemia. Haematologica. 2024 Oct 01; 109(10):3419-3425.
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TRPS1 expression in MPNST is correlated with PRC2 inactivation and loss of H3K27me3. Hum Pathol. 2024 Sep; 151:105632.
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Joint contractures is a recurrent clinical feature of individuals with neurodevelopmental disorder due to FOXP1 likely gene disruptive variants. Am J Med Genet A. 2024 Nov; 194(11):e63713.
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REST-dependent downregulation of von Hippel-Lindau tumor suppressor promotes autophagy in SHH-medulloblastoma. Sci Rep. 2024 06 12; 14(1):13596.
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Atrial Fibrillation and Clonal Hematopoiesis in TET2 and ASXL1. JAMA Cardiol. 2024 Jun 01; 9(6):497-506.
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Repression of Acinetobacter baumannii DNA damage response requires DdrR-assisted binding of UmuDAb dimers to atypical SOS box. J Bacteriol. 2024 06 20; 206(6):e0043223.
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BRAF Mutations in Patients with Myeloid Neoplasms: A Cancer Center Multigene Next-Generation Sequencing Analysis Experience. Int J Mol Sci. 2024 May 09; 25(10).
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Hyperkinetic Movement Disorder Caused by the Recurrent c.892C>T NACC1 Variant. Mov Disord Clin Pract. 2024 Jun; 11(6):708-715.
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Exploring the landscape of somatic ASXL2 mutations in myeloid neoplasms: Frequency and clinical implications. Am J Hematol. 2024 07; 99(7):1434-1436.
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Deep sequencing of candidate genes identified 14 variants associated with smoking abstinence in an ethnically diverse sample. Sci Rep. 2024 03 16; 14(1):6385.