Adaptor Proteins, Signal Transducing
"Adaptor Proteins, Signal Transducing" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A broad category of carrier proteins that play a role in SIGNAL TRANSDUCTION. They generally contain several modular domains, each of which having its own binding activity, and act by forming complexes with other intracellular-signaling molecules. Signal-transducing adaptor proteins lack enzyme activity, however their activity can be modulated by other signal-transducing enzymes
| Descriptor ID |
D048868
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| MeSH Number(s) |
D12.644.360.024 D12.776.157.057 D12.776.476.024
|
| Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Adaptor Proteins, Signal Transducing".
Below are MeSH descriptors whose meaning is more specific than "Adaptor Proteins, Signal Transducing".
This graph shows the total number of publications written about "Adaptor Proteins, Signal Transducing" by people in this website by year, and whether "Adaptor Proteins, Signal Transducing" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1997 | 0 | 2 | 2 |
| 1998 | 0 | 1 | 1 |
| 1999 | 2 | 0 | 2 |
| 2000 | 2 | 3 | 5 |
| 2001 | 5 | 1 | 6 |
| 2002 | 4 | 5 | 9 |
| 2003 | 3 | 7 | 10 |
| 2004 | 4 | 11 | 15 |
| 2005 | 1 | 9 | 10 |
| 2006 | 1 | 6 | 7 |
| 2007 | 7 | 8 | 15 |
| 2008 | 1 | 7 | 8 |
| 2009 | 8 | 3 | 11 |
| 2010 | 9 | 9 | 18 |
| 2011 | 8 | 9 | 17 |
| 2012 | 4 | 10 | 14 |
| 2013 | 10 | 9 | 19 |
| 2014 | 15 | 10 | 25 |
| 2015 | 15 | 15 | 30 |
| 2016 | 13 | 10 | 23 |
| 2017 | 9 | 16 | 25 |
| 2018 | 6 | 8 | 14 |
| 2019 | 5 | 11 | 16 |
| 2020 | 8 | 5 | 13 |
| 2021 | 5 | 6 | 11 |
| 2022 | 2 | 11 | 13 |
| 2023 | 1 | 6 | 7 |
| 2024 | 5 | 6 | 11 |
| 2025 | 11 | 4 | 15 |
| 2026 | 7 | 2 | 9 |
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Below are the most recent publications written about "Adaptor Proteins, Signal Transducing" by people in Profiles.
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Endothelial PERK restricts lymphoid regeneration by reducing DLL4-NOTCH3 signaling at the Pre-B niche. Cell Rep. 2026 Sep 22; 45(9):117953.
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Discovery and Development of First-in-Class Cereblon-Recruiting RIPK1 Degraders. J Med Chem. 2026 Aug 27; 69(16):19822-19840.
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STING is a key driver of Japanese encephalitis virus induced inflammatory response. Commun Biol. 2026 May 30; 9(1).
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Loss of the maternal effect gene NLRP2 impairs embryonic and extra-embryonic development, revealing a novel genetic cause of congenital anomalies?. Biol Reprod. 2026 04 13; 114(4):1469-1485.
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ERK-Mediated Phosphorylation of YAP Defines a Noncanonical FGF Signaling Mechanism in Stem Cells. Adv Sci (Weinh). 2026 Jul; 13(37):e11484.
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TAK1 is a key regulator of oncogenic signaling and differentiation blockade in rhabdomyosarcoma. Oncogene. 2026 May; 45(18):1714-1728.
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Regulation of STK38 by autophagy governs YAP1 activity during paligenosis. Proc Natl Acad Sci U S A. 2026 Apr 07; 123(14):e2517488123.
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YAP Induces a Prorenewal Metabolic State in Cardiomyocytes. Circulation. 2026 Apr 28; 153(17):1296-1313.
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TEAD-independent mechanisms of YAP function in cardiomyocyte cell cycle reentry. Life Sci Alliance. 2026 Apr; 9(4).
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Gene therapy CM-YAPon protects the mouse heart from myocardial infarction. Nat Cardiovasc Res. 2025 Dec; 4(12):1616-1626.