Focal Adhesion Protein-Tyrosine Kinases
"Focal Adhesion Protein-Tyrosine Kinases" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A family of non-receptor, PROLINE-rich protein-tyrosine kinases.
Descriptor ID |
D051416
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MeSH Number(s) |
D08.811.913.696.620.682.725.049 D12.644.360.287 D12.776.476.287
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Concept/Terms |
Focal Adhesion Protein-Tyrosine Kinases- Focal Adhesion Protein-Tyrosine Kinases
- Focal Adhesion Protein Tyrosine Kinases
- Focal Adhesion Protein-Tyrosine Kinase Family
- Focal Adhesion Protein Tyrosine Kinase Family
- Focal Adhesion Kinase
- Focal Adhesion Protein-Tyrosine Kinase
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Below are MeSH descriptors whose meaning is more general than "Focal Adhesion Protein-Tyrosine Kinases".
Below are MeSH descriptors whose meaning is more specific than "Focal Adhesion Protein-Tyrosine Kinases".
This graph shows the total number of publications written about "Focal Adhesion Protein-Tyrosine Kinases" by people in this website by year, and whether "Focal Adhesion Protein-Tyrosine Kinases" was a major or minor topic of these publications.
To see the data from this visualization as text,
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Year | Major Topic | Minor Topic | Total |
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2000 | 0 | 1 | 1 |
2003 | 0 | 1 | 1 |
2004 | 0 | 3 | 3 |
2005 | 0 | 1 | 1 |
2007 | 2 | 0 | 2 |
2010 | 0 | 1 | 1 |
2012 | 0 | 1 | 1 |
2014 | 1 | 0 | 1 |
2016 | 1 | 0 | 1 |
2017 | 1 | 0 | 1 |
2019 | 0 | 1 | 1 |
2022 | 0 | 3 | 3 |
2023 | 0 | 1 | 1 |
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Below are the most recent publications written about "Focal Adhesion Protein-Tyrosine Kinases" by people in Profiles.
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Focal adhesion kinase-YAP signaling axis drives drug-tolerant persister cells and residual disease in lung cancer. Nat Commun. 2024 May 03; 15(1):3741.
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THSD1 Suppresses Autophagy-Mediated Focal Adhesion Turnover by Modulating the FAK-Beclin 1 Pathway. Int J Mol Sci. 2024 Feb 10; 25(4).
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Integrin signaling is critical for myeloid-mediated support of T-cell acute lymphoblastic leukemia. Nat Commun. 2023 10 07; 14(1):6270.
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Loss of non-motor kinesin KIF26A causes congenital brain malformations via dysregulated neuronal migration and axonal growth as well as apoptosis. Dev Cell. 2022 10 24; 57(20):2381-2396.e13.
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Biliverdin reductase bridges focal adhesion kinase to Src to modulate synaptic signaling. Sci Signal. 2022 05 10; 15(733):eabh3066.
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EZH2 engages TGF? signaling to promote breast cancer bone metastasis via integrin ?1-FAK activation. Nat Commun. 2022 05 10; 13(1):2543.
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GSDMB is increased in IBD and regulates epithelial restitution/repair independent of pyroptosis. Cell. 2022 01 20; 185(2):283-298.e17.
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Disturbed flow-induced FAK K152 SUMOylation initiates the formation of pro-inflammation positive feedback loop by inducing reactive oxygen species production in endothelial cells. Free Radic Biol Med. 2021 12; 177:404-418.
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Pancreatic cancer stroma: an update on therapeutic targeting strategies. Nat Rev Gastroenterol Hepatol. 2020 08; 17(8):487-505.
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Detecting Protein-Protein Interaction Based on Protein Fragment Complementation Assay. Curr Protein Pept Sci. 2020; 21(6):598-610.