Proto-Oncogene Proteins c-maf
"Proto-Oncogene Proteins c-maf" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Maf proto-oncogene protein is the major cellular homolog of the V-MAF ONCOGENE PROTEIN. It was the first of the mammalian MAF TRANSCRIPTION FACTORS identified, and it is induced in activated T-LYMPHOCYTES and regulates GENETIC TRANSCRIPTION of INTERLEUKIN-4. c-maf is frequently translocated to an immunoglobulin locus in MULTIPLE MYELOMA.
| Descriptor ID |
D051257
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| MeSH Number(s) |
D12.776.260.108.500.061.750 D12.776.624.664.700.184 D12.776.930.127.500.061.750
|
| Concept/Terms |
Proto-Oncogene Proteins c-maf- Proto-Oncogene Proteins c-maf
- Proteins c-maf, Proto-Oncogene
- Proto Oncogene Proteins c maf
- c-maf, Proto-Oncogene Proteins
- c-maf Proto-Oncogene Protein
- Proto-Oncogene Protein, c-maf
- Proto-Oncogene Protein c-maf
- Proto Oncogene Protein c maf
- c-maf, Proto-Oncogene Protein
- c-maf Protein
- maf Proto-Oncogene Protein
- Proto-Oncogene Protein, maf
- maf Proto Oncogene Protein
|
Below are MeSH descriptors whose meaning is more general than "Proto-Oncogene Proteins c-maf".
Below are MeSH descriptors whose meaning is more specific than "Proto-Oncogene Proteins c-maf".
This graph shows the total number of publications written about "Proto-Oncogene Proteins c-maf" by people in this website by year, and whether "Proto-Oncogene Proteins c-maf" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2000 | 0 | 1 | 1 |
| 2001 | 0 | 1 | 1 |
| 2002 | 0 | 2 | 2 |
| 2004 | 0 | 1 | 1 |
| 2007 | 0 | 1 | 1 |
| 2010 | 0 | 1 | 1 |
| 2015 | 1 | 0 | 1 |
| 2019 | 0 | 1 | 1 |
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Below are the most recent publications written about "Proto-Oncogene Proteins c-maf" by people in Profiles.
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The SAGA acetyltransferase module is required for the maintenance of MAF and MYC oncogenic gene expression programs in multiple myeloma. Genes Dev. 2024 Sep 19; 38(15-16):738-754.
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Characterization of Transcriptional Regulatory Networks that Promote and Restrict Identities and Functions of Intestinal Innate Lymphoid Cells. Immunity. 2019 07 16; 51(1):185-197.e6.
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Effect of MAF amplification on treatment outcomes with adjuvant zoledronic acid in early breast cancer: a secondary analysis of the international, open-label, randomised, controlled, phase 3 AZURE (BIG 01/04) trial. Lancet Oncol. 2017 11; 18(11):1543-1552.
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Enhanced MAF Oncogene Expression and Breast Cancer Bone Metastasis. J Natl Cancer Inst. 2015 Dec; 107(12):djv256.
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APOBEC family mutational signatures are associated with poor prognosis translocations in multiple myeloma. Nat Commun. 2015 Apr 23; 6:6997.
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Mutations Impairing GSK3-Mediated MAF Phosphorylation Cause Cataract, Deafness, Intellectual Disability, Seizures, and a Down Syndrome-like Facies. Am J Hum Genet. 2015 May 07; 96(5):816-25.
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Activation of the transcription factor c-Maf in T cells is dependent on the CARMA1-IKK? signaling cascade. Sci Signal. 2013 Dec 17; 6(306):ra110.
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Transcriptional regulation of follicular T-helper (Tfh) cells. Immunol Rev. 2013 Mar; 252(1):139-45.
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Lens fiber cell differentiation and denucleation are disrupted through expression of the N-terminal nuclear receptor box of NCOA6 and result in p53-dependent and p53-independent apoptosis. Mol Biol Cell. 2010 Jul 15; 21(14):2453-68.
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IL-25 augments type 2 immune responses by enhancing the expansion and functions of TSLP-DC-activated Th2 memory cells. J Exp Med. 2007 Aug 06; 204(8):1837-47.