Nuclear Matrix-Associated Proteins
"Nuclear Matrix-Associated Proteins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A broad category of nuclear proteins that are components of or participate in the formation of the NUCLEAR MATRIX.
| Descriptor ID |
D034521
|
| MeSH Number(s) |
D12.776.660.650
|
| Concept/Terms |
Nuclear Matrix-Associated Proteins- Nuclear Matrix-Associated Proteins
- Matrix-Associated Proteins, Nuclear
- Nuclear Matrix Associated Proteins
- Protein, Nuclear Scaffold
- Nuclear Scaffold Protein
- Scaffold Protein, Nuclear
- Nuclear Matrix Proteins
- Matrix Proteins, Nuclear
- Nuclear Scaffold Proteins
- Scaffold Proteins, Nuclear
|
Below are MeSH descriptors whose meaning is more general than "Nuclear Matrix-Associated Proteins".
Below are MeSH descriptors whose meaning is more specific than "Nuclear Matrix-Associated Proteins".
This graph shows the total number of publications written about "Nuclear Matrix-Associated Proteins" by people in this website by year, and whether "Nuclear Matrix-Associated Proteins" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1995 | 0 | 1 | 1 |
| 1997 | 1 | 0 | 1 |
| 2000 | 2 | 0 | 2 |
| 2003 | 0 | 1 | 1 |
| 2005 | 1 | 1 | 2 |
| 2008 | 1 | 0 | 1 |
| 2009 | 2 | 0 | 2 |
| 2015 | 1 | 0 | 1 |
| 2016 | 1 | 0 | 1 |
| 2024 | 0 | 1 | 1 |
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Below are the most recent publications written about "Nuclear Matrix-Associated Proteins" by people in Profiles.
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MATR3 pathogenic variants differentially impair its cryptic splicing repression function. FEBS Lett. 2024 Feb; 598(4):415-436.
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CARM1 and Paraspeckles Regulate Pre-implantation Mouse Embryo Development. Cell. 2018 12 13; 175(7):1902-1916.e13.
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Abro1 maintains genome stability and limits replication stress by protecting replication fork stability. Genes Dev. 2017 07 15; 31(14):1469-1482.
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Induction of NKG2D Ligands on Solid Tumors Requires Tumor-Specific CD8+ T Cells and Histone Acetyltransferases. Cancer Immunol Res. 2017 04; 5(4):300-311.
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Congenital heart defects and left ventricular non-compaction in males with loss-of-function variants in NONO. J Med Genet. 2017 01; 54(1):47-53.
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Higher-Order Assembly of BRCC36-KIAA0157 Is Required for DUB Activity and Biological Function. Mol Cell. 2015 Sep 17; 59(6):970-83.
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IL-30 (IL27p28) attenuates liver fibrosis through inducing NKG2D-rae1 interaction between NKT and activated hepatic stellate cells in mice. Hepatology. 2014 Dec; 60(6):2027-39.
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Assessment of autoantibodies to meningioma in a population-based study. Am J Epidemiol. 2013 Jan 01; 177(1):75-83.
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Organization of the ENaC-regulatory machinery. Crit Rev Biochem Mol Biol. 2012 Jul-Aug; 47(4):349-59.
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RAE-1, a novel PHR binding protein, is required for axon termination and synapse formation in Caenorhabditis elegans. J Neurosci. 2012 Feb 22; 32(8):2628-36.