"Tumor Necrosis Factors" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A family of proteins that were originally identified by their ability to cause NECROSIS of NEOPLASMS. Their necrotic effect on cells is mediated through TUMOR NECROSIS FACTOR RECEPTORS which induce APOPTOSIS.
| Descriptor ID |
D048069
|
| MeSH Number(s) |
D12.644.276.374.750 D12.776.467.374.750 D23.529.374.750
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Tumor Necrosis Factors".
Below are MeSH descriptors whose meaning is more specific than "Tumor Necrosis Factors".
This graph shows the total number of publications written about "Tumor Necrosis Factors" by people in this website by year, and whether "Tumor Necrosis Factors" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2004 | 1 | 0 | 1 |
| 2008 | 1 | 0 | 1 |
| 2010 | 0 | 1 | 1 |
| 2013 | 0 | 1 | 1 |
| 2014 | 0 | 1 | 1 |
| 2017 | 1 | 0 | 1 |
| 2023 | 0 | 1 | 1 |
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Below are the most recent publications written about "Tumor Necrosis Factors" by people in Profiles.
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TSLP and downstream molecules in experimental mouse allergic conjunctivitis. Invest Ophthalmol Vis Sci. 2010 Jun; 51(6):3076-82.
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Plasmacytoid dendritic cells induce NK cell-dependent, tumor antigen-specific T cell cross-priming and tumor regression in mice. J Clin Invest. 2008 Mar; 118(3):1165-75.
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Hyperthermia engages the intrinsic apoptotic pathway by enhancing upstream caspase activation to overcome apoptotic resistance in MCF-7 breast adenocarcinoma cells. J Cell Biochem. 2006 May 15; 98(2):356-69.
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Stat1 deficiency in the host enhances interleukin-12-mediated tumor regression. Cancer Res. 2006 Apr 15; 66(8):4461-7.
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Polymorphisms of the FAS and FAS ligand genes associated with risk of cutaneous malignant melanoma. Pharmacogenet Genomics. 2006 Apr; 16(4):253-63.
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Inhibition of the phosphatidylinositol-3 kinase/Akt promotes G1 cell cycle arrest and apoptosis in Hodgkin lymphoma. Br J Haematol. 2006 Feb; 132(4):503-11.