"Oxidoreductases" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The class of all enzymes catalyzing oxidoreduction reactions. The substrate that is oxidized is regarded as a hydrogen donor. The systematic name is based on donor:acceptor oxidoreductase. The recommended name will be dehydrogenase, wherever this is possible; as an alternative, reductase can be used. Oxidase is only used in cases where O2 is the acceptor. (Enzyme Nomenclature, 1992, p9)
| Descriptor ID |
D010088
|
| MeSH Number(s) |
D08.811.682
|
| Concept/Terms |
Oxidoreductases- Oxidoreductases
- Reductases
- Reductase
- Dehydrogenase
- Dehydrogenases
|
Below are MeSH descriptors whose meaning is more general than "Oxidoreductases".
Below are MeSH descriptors whose meaning is more specific than "Oxidoreductases".
This graph shows the total number of publications written about "Oxidoreductases" by people in this website by year, and whether "Oxidoreductases" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1995 | 1 | 0 | 1 |
| 1997 | 0 | 1 | 1 |
| 1998 | 4 | 0 | 4 |
| 1999 | 4 | 1 | 5 |
| 2000 | 0 | 2 | 2 |
| 2001 | 2 | 2 | 4 |
| 2002 | 3 | 0 | 3 |
| 2003 | 1 | 2 | 3 |
| 2004 | 2 | 2 | 4 |
| 2005 | 9 | 3 | 12 |
| 2006 | 3 | 0 | 3 |
| 2007 | 4 | 2 | 6 |
| 2008 | 1 | 0 | 1 |
| 2009 | 5 | 3 | 8 |
| 2010 | 0 | 1 | 1 |
| 2011 | 2 | 1 | 3 |
| 2012 | 1 | 1 | 2 |
| 2013 | 4 | 0 | 4 |
| 2014 | 4 | 0 | 4 |
| 2015 | 2 | 3 | 5 |
| 2016 | 3 | 1 | 4 |
| 2017 | 0 | 3 | 3 |
| 2018 | 0 | 3 | 3 |
| 2019 | 1 | 0 | 1 |
| 2020 | 1 | 2 | 3 |
| 2021 | 0 | 2 | 2 |
| 2022 | 2 | 1 | 3 |
| 2023 | 0 | 4 | 4 |
| 2024 | 0 | 1 | 1 |
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Below are the most recent publications written about "Oxidoreductases" by people in Profiles.
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NADPH oxidase overexpression and mitochondrial OxPhos impairment are more profound in human hearts donated after circulatory death than brain death. Am J Physiol Heart Circ Physiol. 2024 03 01; 326(3):H548-H562.
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Growth requirement for methionine in human melanoma-derived cell lines with different levels of MMACHC expression and methylation. Mol Genet Metab. 2024 Jan; 141(1):108111.
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Mechanism of stepwise electron transfer in six-transmembrane epithelial antigen of the prostate (STEAP) 1 and 2. Elife. 2023 Nov 20; 12.
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Heterozygous nonsense variants in the ferritin heavy-chain gene FTH1 cause a neuroferritinopathy. HGG Adv. 2023 Oct 12; 4(4):100236.
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Ceramide compensation by ceramide synthases preserves retinal function and structure in a retinal dystrophy mouse model. Dis Model Mech. 2023 07 01; 16(7).
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Bi-allelic variants in HMGCR cause an autosomal-recessive progressive limb-girdle muscular dystrophy. Am J Hum Genet. 2023 06 01; 110(6):989-997.
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Applying HT-SAXS to chemical ligand screening. Methods Enzymol. 2023; 678:331-350.
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Transmembrane helices mediate the formation of a stable ternary complex of b5R, cyt b5, and SCD1. Commun Biol. 2022 09 12; 5(1):956.
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Mutations in Hcfc1 and Ronin result in an inborn error of cobalamin metabolism and ribosomopathy. Nat Commun. 2022 01 10; 13(1):134.
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Gastric epithelial attachment of Helicobacter pylori induces EphA2 and NMHC-IIA receptors for Epstein-Barr virus. Cancer Sci. 2021 Nov; 112(11):4799-4811.