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Loss of biphasic effect on Na/K-ATPase activity by angiotensin II involves defective angiotensin type 1 receptor-nitric oxide signaling.
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Loss of biphasic effect on Na/K-ATPase activity by angiotensin II involves defective angiotensin type 1 receptor-nitric oxide signaling.
Loss of biphasic effect on Na/K-ATPase activity by angiotensin II involves defective angiotensin type 1 receptor-nitric oxide signaling. Hypertension. 2008 Dec; 52(6):1099-105.
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PubMed
subject areas
Angiotensin II
Animals
Antihypertensive Agents
Antioxidants
Benzimidazoles
Biphenyl Compounds
Blood Pressure
Buthionine Sulfoximine
Cyclic GMP
Cyclic N-Oxides
Enzyme Inhibitors
Focal Adhesion Kinase 2
Hypertension, Renal
Kidney Tubules, Proximal
Male
Mitogen-Activated Protein Kinases
Nitric Oxide
Oxidative Stress
Rats
Rats, Sprague-Dawley
Receptor, Angiotensin, Type 1
Signal Transduction
Sodium-Hydrogen Exchanger 3
Sodium-Hydrogen Exchangers
Sodium-Potassium-Exchanging ATPase
Spin Labels
Tetrazoles
Vasoconstrictor Agents
authors with profiles
MUSTAFA F. LOKHANDWALA
ANEES AHMAD BANDAY