Excitation Contraction Coupling
"Excitation Contraction Coupling" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A process fundamental to muscle physiology whereby an electrical stimulus or action potential triggers a myocyte to depolarize and contract. This mechanical muscle contraction response is regulated by entry of calcium ions into the cell.
Descriptor ID |
D056966
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MeSH Number(s) |
G02.111.820.800.100.500 G04.835.199 G11.427.494.235
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Concept/Terms |
Excitation Contraction Coupling- Excitation Contraction Coupling
- Contraction Coupling, Excitation
- Contraction Couplings, Excitation
- Coupling, Excitation Contraction
- Couplings, Excitation Contraction
- Excitation Contraction Couplings
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Below are MeSH descriptors whose meaning is more general than "Excitation Contraction Coupling".
Below are MeSH descriptors whose meaning is more specific than "Excitation Contraction Coupling".
This graph shows the total number of publications written about "Excitation Contraction Coupling" by people in this website by year, and whether "Excitation Contraction Coupling" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2010 | 1 | 2 | 3 |
2011 | 0 | 1 | 1 |
2012 | 1 | 1 | 2 |
2013 | 1 | 0 | 1 |
2014 | 0 | 2 | 2 |
2017 | 1 | 1 | 2 |
2021 | 1 | 0 | 1 |
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Below are the most recent publications written about "Excitation Contraction Coupling" by people in Profiles.
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SPEG: a key regulator of cardiac calcium homeostasis. Cardiovasc Res. 2021 08 29; 117(10):2175-2185.
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Force development and intracellular Ca2+ in intact cardiac muscles from gravin mutant mice. Eur J Pharmacol. 2017 Jul 15; 807:117-126.
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Sheet-Like Remodeling of the Transverse Tubular System in Human Heart Failure Impairs Excitation-Contraction Coupling and Functional Recovery by Mechanical Unloading. Circulation. 2017 Apr 25; 135(17):1632-1645.
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Emerging roles of junctophilin-2 in the heart and implications for cardiac diseases. Cardiovasc Res. 2014 Jul 15; 103(2):198-205.
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Microtubule-mediated defects in junctophilin-2 trafficking contribute to myocyte transverse-tubule remodeling and Ca2+ handling dysfunction in heart failure. Circulation. 2014 Apr 29; 129(17):1742-50.
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Critical roles of junctophilin-2 in T-tubule and excitation-contraction coupling maturation during postnatal development. Cardiovasc Res. 2013 Oct 01; 100(1):54-62.
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Store-operated Ca?? entry is not required for store refilling in skeletal muscle. Clin Exp Pharmacol Physiol. 2013 May; 40(5):338-44.
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Cardiac ryanodine receptors control heart rate and rhythmicity in adult mice. Cardiovasc Res. 2012 Dec 01; 96(3):372-80.
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Cardiac G-protein-coupled receptor kinase 2 ablation induces a novel Ca2+ handling phenotype resistant to adverse alterations and remodeling after myocardial infarction. Circulation. 2012 May 01; 125(17):2108-18.
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Proarrhythmic atrial calcium cycling in the diseased heart. Adv Exp Med Biol. 2012; 740:1175-91.