"Myocytes, Cardiac" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Striated muscle cells found in the heart. They are derived from cardiac myoblasts (MYOBLASTS, CARDIAC).
| Descriptor ID |
D032383
|
| MeSH Number(s) |
A07.541.704.570 A10.690.552.750.570 A11.620.500
|
| Concept/Terms |
Myocytes, Cardiac- Myocytes, Cardiac
- Cardiac Myocyte
- Cardiac Myocytes
- Myocyte, Cardiac
- Muscle Cells, Heart
- Cell, Heart Muscle
- Cells, Heart Muscle
- Heart Muscle Cell
- Heart Muscle Cells
- Muscle Cell, Heart
- Cardiomyocytes
- Cardiomyocyte
- Muscle Cells, Cardiac
- Cardiac Muscle Cell
- Cardiac Muscle Cells
- Cell, Cardiac Muscle
- Cells, Cardiac Muscle
- Muscle Cell, Cardiac
|
Below are MeSH descriptors whose meaning is more general than "Myocytes, Cardiac".
Below are MeSH descriptors whose meaning is more specific than "Myocytes, Cardiac".
This graph shows the total number of publications written about "Myocytes, Cardiac" by people in this website by year, and whether "Myocytes, Cardiac" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2002 | 0 | 2 | 2 |
| 2003 | 5 | 2 | 7 |
| 2004 | 7 | 5 | 12 |
| 2005 | 3 | 5 | 8 |
| 2006 | 3 | 5 | 8 |
| 2007 | 13 | 8 | 21 |
| 2008 | 17 | 8 | 25 |
| 2009 | 6 | 6 | 12 |
| 2010 | 8 | 8 | 16 |
| 2011 | 10 | 9 | 19 |
| 2012 | 4 | 8 | 12 |
| 2013 | 11 | 7 | 18 |
| 2014 | 19 | 2 | 21 |
| 2015 | 19 | 8 | 27 |
| 2016 | 18 | 12 | 30 |
| 2017 | 20 | 7 | 27 |
| 2018 | 18 | 7 | 25 |
| 2019 | 15 | 10 | 25 |
| 2020 | 18 | 9 | 27 |
| 2021 | 18 | 14 | 32 |
| 2022 | 4 | 14 | 18 |
| 2023 | 7 | 14 | 21 |
| 2024 | 18 | 8 | 26 |
| 2025 | 9 | 7 | 16 |
| 2026 | 3 | 2 | 5 |
To return to the timeline,
click here.
Below are the most recent publications written about "Myocytes, Cardiac" by people in Profiles.
-
Nitric oxide supplementation during extracorporeal resuscitation drives oxidative-inflammatory signaling and metabolic suppression in the post-cardiac arrest heart. Mol Med. 2026 May 08; 32(1).
-
Phospho-mimic ?III-tubulin rescues microtubule and cardiac defects in Duchenne muscular dystrophy mice. J Mol Cell Cardiol. 2026 Jun; 215:6-19.
-
YAP Induces a Prorenewal Metabolic State in Cardiomyocytes. Circulation. 2026 Apr 28; 153(17):1296-1313.
-
Ventricular assist device unloading reverses microvascular senescence in single ventricle disease. Nat Cardiovasc Res. 2026 Mar; 5(3):262-280.
-
TEAD-independent mechanisms of YAP function in cardiomyocyte cell cycle reentry. Life Sci Alliance. 2026 Apr; 9(4).
-
Cyclic AMP-dependent regulation of ryanodine receptors in healthy and diseased hearts. J Mol Cell Cardiol. 2026 Feb; 211:53-62.
-
RRAD-reduction reveals efficacy of targeting L-type calcium channel regulation for treatment of heart failure. Cardiovasc Res. 2025 Nov 22; 121(14):2204-2221.
-
Cardiomyocyte maturation and proliferation is a flip coin. BMC Cardiovasc Disord. 2025 11 21; 25(1):880.
-
Gene therapy CM-YAPon protects the mouse heart from myocardial infarction. Nat Cardiovasc Res. 2025 Dec; 4(12):1616-1626.
-
Fibroblasts Are the Primary Contributors to a Disrupted Micro-Environment in End-Stage Pediatric Hypertrophic Cardiomyopathy. Circ Genom Precis Med. 2025 Dec; 18(6):e005192.