"Acute-Phase Proteins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Proteins that are secreted into the blood in increased or decreased quantities by hepatocytes in response to trauma, inflammation, or disease. These proteins can serve as inhibitors or mediators of the inflammatory processes. Certain acute-phase proteins have been used to diagnose and follow the course of diseases or as tumor markers.
| Descriptor ID |
D000209
|
| MeSH Number(s) |
D12.776.124.050
|
| Concept/Terms |
Acute-Phase Proteins- Acute-Phase Proteins
- Acute Phase Proteins
- Proteins, Acute-Phase
- Proteins, Acute Phase
- Reactants, Acute-Phase
- Reactants, Acute Phase
- Acute-Phase Reactants
- Acute Phase Reactants
- Acute-Phase Protein
- Acute Phase Protein
- Protein, Acute-Phase
|
Below are MeSH descriptors whose meaning is more general than "Acute-Phase Proteins".
Below are MeSH descriptors whose meaning is more specific than "Acute-Phase Proteins".
This graph shows the total number of publications written about "Acute-Phase Proteins" by people in this website by year, and whether "Acute-Phase Proteins" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1996 | 1 | 0 | 1 |
| 1998 | 1 | 1 | 2 |
| 1999 | 1 | 1 | 2 |
| 2000 | 1 | 0 | 1 |
| 2001 | 1 | 1 | 2 |
| 2002 | 1 | 2 | 3 |
| 2003 | 0 | 2 | 2 |
| 2004 | 1 | 1 | 2 |
| 2005 | 1 | 1 | 2 |
| 2007 | 1 | 1 | 2 |
| 2008 | 1 | 0 | 1 |
| 2009 | 2 | 0 | 2 |
| 2011 | 1 | 1 | 2 |
| 2012 | 1 | 1 | 2 |
| 2013 | 1 | 1 | 2 |
| 2014 | 1 | 3 | 4 |
| 2015 | 1 | 1 | 2 |
| 2017 | 0 | 1 | 1 |
| 2022 | 0 | 2 | 2 |
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click here.
Below are the most recent publications written about "Acute-Phase Proteins" by people in Profiles.
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Long-term Use of Proton Pump Inhibitors Is Associated With Increased Microbial Product Translocation, Innate Immune Activation, and Reduced Immunologic Recovery in Patients With Chronic Human Immunodeficiency Virus-1 Infection. Clin Infect Dis. 2017 Oct 30; 65(10):1638-1643.
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A pilot study to evaluate renal hemodynamics in cirrhosis by simultaneous glomerular filtration rate, renal plasma flow, renal resistive indices and biomarkers measurements. Am J Nephrol. 2014; 39(6):543-52.
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Role of innate immunity and altered intestinal motility in LPS- and MnCl2-induced intestinal intussusception in mice. Am J Physiol Gastrointest Liver Physiol. 2014 Mar 01; 306(5):G445-53.
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Clinical and biomarker outcomes of the phase II vandetanib study from the BATTLE trial. J Thorac Oncol. 2013 May; 8(5):658-61.
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Epidermal growth factor down-regulates the expression of neutrophil gelatinase-associated lipocalin (NGAL) through E-cadherin in pancreatic cancer cells. Cancer. 2011 Jun 01; 117(11):2408-18.
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Inhibition of the proliferation and invasion of hepatocellular carcinoma cells by lipocalin 2 through blockade of JNK and PI3K/Akt signaling. Int J Oncol. 2011 Feb; 38(2):325-33.
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Neutrophil gelatinase-associated lipocalin: a novel suppressor of invasion and angiogenesis in pancreatic cancer. Cancer Res. 2008 Aug 01; 68(15):6100-8.
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Lipocalin 2 is required for BCR-ABL-induced tumorigenesis. Oncogene. 2008 Oct 16; 27(47):6110-9.
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Urine neutrophil gelatinase-associated lipocalin is an early marker of acute kidney injury in critically ill children: a prospective cohort study. Crit Care. 2007; 11(4):R84.
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Plasma protein profiling for diagnosis of pancreatic cancer reveals the presence of host response proteins. Clin Cancer Res. 2005 Feb 01; 11(3):1110-8.