"snRNP Core Proteins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The protein components that constitute the common core of small nuclear ribonucleoprotein particles. These proteins are commonly referred as Sm nuclear antigens due to their antigenic nature.
| Descriptor ID |
D055517
|
| MeSH Number(s) |
D12.776.157.725.500.875.700 D12.776.660.625.750 D12.776.664.962.500.875.700 D23.050.290.875 D23.050.422.750
|
| Concept/Terms |
snRNP Core Proteins- snRNP Core Proteins
- Core Proteins, snRNP
- Sm Core Proteins
- Core Proteins, Sm
- Sm Nuclear Antigens
- Antigens, Sm Nuclear
- Nuclear Antigens, Sm
- Small Nuclear Ribonucleoprotein Core Proteins
Small Nuclear Ribonucleoprotein-Associated Protein D1- Small Nuclear Ribonucleoprotein-Associated Protein D1
- Small Nuclear Ribonucleoprotein Associated Protein D1
- SmD Autoantigen
- Autoantigen, SmD
- snRNP Core Protein D1
- SmD Antigen
- Antigen, SmD
- Small Nuclear Ribonucleoprotein D1 Polypeptide 16kDa
- Small Nuclear Ribonucleoprotein Sm D1
|
Below are MeSH descriptors whose meaning is more general than "snRNP Core Proteins".
Below are MeSH descriptors whose meaning is more specific than "snRNP Core Proteins".
This graph shows the total number of publications written about "snRNP Core Proteins" by people in this website by year, and whether "snRNP Core Proteins" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1997 | 0 | 1 | 1 |
| 2006 | 0 | 1 | 1 |
| 2007 | 0 | 1 | 1 |
| 2008 | 0 | 1 | 1 |
| 2012 | 0 | 2 | 2 |
| 2018 | 1 | 0 | 1 |
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Below are the most recent publications written about "snRNP Core Proteins" by people in Profiles.
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snRPN controls the ability of neurons to regenerate axons. Restor Neurol Neurosci. 2018; 36(1):31-43.
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Evolutionarily conserved protein ERH controls CENP-E mRNA splicing and is required for the survival of KRAS mutant cancer cells. Proc Natl Acad Sci U S A. 2012 Dec 26; 109(52):E3659-67.
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Ube3a-ATS is an atypical RNA polymerase II transcript that represses the paternal expression of Ube3a. Hum Mol Genet. 2012 Jul 01; 21(13):3001-12.
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An unexpected function of the Prader-Willi syndrome imprinting center in maternal imprinting in mice. PLoS One. 2012; 7(4):e34348.
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Prader-Willi phenotype caused by paternal deficiency for the HBII-85 C/D box small nucleolar RNA cluster. Nat Genet. 2008 Jun; 40(6):719-21.
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Hfq structure, function and ligand binding. Curr Opin Microbiol. 2007 Apr; 10(2):125-33.
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Minimal phenotype in a girl with trisomy 15q due to t(X;15)(q22.3;q11.2) translocation. Am J Med Genet A. 2006 Mar 01; 140(5):442-52.
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Mouse imprinting defect mutations that model Angelman syndrome. Genesis. 2006 Jan; 44(1):12-22.
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A mouse model for Prader-Willi syndrome imprinting-centre mutations. Nat Genet. 1998 May; 19(1):25-31.
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A candidate model for Angelman syndrome in the mouse. Mamm Genome. 1997 Jul; 8(7):472-8.