"Podocytes" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Highly differentiated epithelial cells of the visceral layer of BOWMAN CAPSULE of the KIDNEY. They are composed of a cell body with major CELL SURFACE EXTENSIONS and secondary fingerlike extensions called pedicels. They enwrap the KIDNEY GLOMERULUS capillaries with their cell surface extensions forming a filtration structure. The pedicels of neighboring podocytes interdigitate with each other leaving between them filtration slits that are bridged by an extracellular structure impermeable to large macromolecules called the slit diaphragm, and provide the last barrier to protein loss in the KIDNEY.
| Descriptor ID |
D050199
|
| MeSH Number(s) |
A05.810.453.324.359.372.650 A05.810.453.736.520.720 A11.436.720
|
| Concept/Terms |
Podocytes- Podocytes
- Podocyte
- Glomerular Visceral Epithelial Cells
- Visceral Epithelial Cells, Glomerular
- Epithelial Cells, Glomerular Visceral
|
Below are MeSH descriptors whose meaning is more general than "Podocytes".
Below are MeSH descriptors whose meaning is more specific than "Podocytes".
This graph shows the total number of publications written about "Podocytes" by people in this website by year, and whether "Podocytes" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2006 | 2 | 0 | 2 |
| 2007 | 2 | 0 | 2 |
| 2008 | 3 | 0 | 3 |
| 2009 | 2 | 1 | 3 |
| 2010 | 4 | 1 | 5 |
| 2011 | 2 | 2 | 4 |
| 2012 | 6 | 1 | 7 |
| 2013 | 4 | 1 | 5 |
| 2014 | 4 | 1 | 5 |
| 2015 | 1 | 1 | 2 |
| 2016 | 4 | 1 | 5 |
| 2017 | 0 | 3 | 3 |
| 2018 | 2 | 0 | 2 |
| 2019 | 3 | 3 | 6 |
| 2020 | 2 | 3 | 5 |
| 2021 | 2 | 1 | 3 |
| 2022 | 4 | 0 | 4 |
| 2023 | 3 | 0 | 3 |
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Below are the most recent publications written about "Podocytes" by people in Profiles.
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Podocyte cell-specific Npr1 is required for blood pressure and renal homeostasis in male and female mice: role of sex-specific differences. Physiol Genomics. 2024 Oct 01; 56(10):672-690.
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Genetic reprogramming with stem cells regenerates glomerular epithelial podocytes in Alport syndrome. Life Sci Alliance. 2024 Jun; 7(6).
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Contribution of membrane raft redox signalling to visfatin-induced inflammasome activation and podocyte injury. Aging (Albany NY). 2023 11 17; 15(22):12738-12748.
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Irisin ameliorates diabetic kidney disease by restoring autophagy in podocytes. FASEB J. 2023 10; 37(10):e23175.
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No NLRP3 inflammasome activity in kidney epithelial cells, not even when the NLRP3-A350V Muckle-Wells variant is expressed in podocytes of diabetic mice. Front Immunol. 2023; 14:1230050.
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Podocyte derived TNF-a mediates monocyte differentiation and contributes to glomerular injury. FASEB J. 2022 12; 36(12):e22622.
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Growth hormone induces transforming growth factor-?1 in podocytes: Implications in podocytopathy and proteinuria. Biochim Biophys Acta Mol Cell Res. 2023 02; 1870(2):119391.
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Integrative transcriptome analysis reveals TEKT2 and PIAS2 involvement in diabetic nephropathy. FASEB J. 2022 11; 36(11):e22592.
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Protecting glomerulus: role of angiotensin-II type 2 receptor. Clin Sci (Lond). 2022 10 28; 136(20):1467-1470.
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The key role of NLRP3 and STING in APOL1-associated podocytopathy. J Clin Invest. 2021 10 15; 131(20).