Potassium Channels, Calcium-Activated
"Potassium Channels, Calcium-Activated" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Potassium channels whose activation is dependent on intracellular calcium concentrations.
Descriptor ID |
D024681
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MeSH Number(s) |
D12.776.157.530.400.600.150 D12.776.543.550.450.750.150 D12.776.543.585.400.750.150
|
Concept/Terms |
Potassium Channels, Calcium-Activated- Potassium Channels, Calcium-Activated
- Potassium Channels, Calcium Activated
- Calcium-Activated Potassium Channels
- Calcium Activated Potassium Channels
- K+ Channels, Ca2+-Activated
- K+ Channels, Ca2+ Activated
- Potassium Channels, Calcium-Dependent
- Potassium Channels, Calcium Dependent
- Potassium Channel, Calcium-Activated
- Potassium Channel, Calcium Activated
- Calcium-Activated Potassium Channel
- Calcium Activated Potassium Channel
- Calcium-Dependent Potassium Channels
- Calcium Dependent Potassium Channels
- Channels, Calcium-Dependent Potassium
- K+ Channels, Calcium-Activated
- Calcium-Activated K+ Channels
- K+ Channels, Calcium Activated
- Ca2+-Activated K+ Channels
- Ca2+ Activated K+ Channels
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Below are MeSH descriptors whose meaning is more general than "Potassium Channels, Calcium-Activated".
Below are MeSH descriptors whose meaning is more specific than "Potassium Channels, Calcium-Activated".
This graph shows the total number of publications written about "Potassium Channels, Calcium-Activated" by people in this website by year, and whether "Potassium Channels, Calcium-Activated" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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1999 | 2 | 0 | 2 |
2001 | 1 | 0 | 1 |
2002 | 1 | 0 | 1 |
2003 | 1 | 0 | 1 |
2004 | 1 | 1 | 2 |
2005 | 2 | 1 | 3 |
2006 | 1 | 0 | 1 |
2007 | 1 | 0 | 1 |
2010 | 1 | 0 | 1 |
2014 | 1 | 0 | 1 |
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Below are the most recent publications written about "Potassium Channels, Calcium-Activated" by people in Profiles.
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Inhibition of Small-Conductance Ca2+-Activated K+ Channels: The Long-Awaited Breakthrough for Antiarrhythmic Drug Therapy of Atrial Fibrillation? Circ Arrhythm Electrophysiol. 2017 10; 10(10).
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Protein kinase CK2 contributes to diminished small conductance Ca2+-activated K+ channel activity of hypothalamic pre-sympathetic neurons in hypertension. J Neurochem. 2014 Sep; 130(5):657-67.
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Endothelium-dependent hyperpolarization: out of the dish and into the brain. J Cereb Blood Flow Metab. 2011 May; 31(5):1173-4.
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TRPC6 channels and their binding partners in podocytes: role in glomerular filtration and pathophysiology. Am J Physiol Renal Physiol. 2010 Oct; 299(4):F689-701.
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Neph1 regulates steady-state surface expression of Slo1 Ca(2+)-activated K(+) channels: different effects in embryonic neurons and podocytes. Am J Physiol Cell Physiol. 2009 Dec; 297(6):C1379-88.
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The beta 1 subunit of L-type voltage-gated Ca2+ channels independently binds to and inhibits the gating of large-conductance Ca2+-activated K+ channels. Mol Pharmacol. 2008 Feb; 73(2):369-78.
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A novel actin-binding domain on Slo1 calcium-activated potassium channels is necessary for their expression in the plasma membrane. Mol Pharmacol. 2008 Feb; 73(2):359-68.
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Electrophysiological properties of human adipose tissue-derived stem cells. Am J Physiol Cell Physiol. 2007 Nov; 293(5):C1539-50.
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Endothelium-independent relaxation by adrenomedullin in pregnant rat mesenteric artery: role of cAMP-dependent protein kinase A and calcium-activated potassium channels. J Pharmacol Exp Ther. 2006 Jun; 317(3):1269-75.
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The p38 mitogen-activated protein kinase pathway negatively regulates Ca2+-activated K+ channel trafficking in developing parasympathetic neurons. J Neurochem. 2005 Jul; 94(2):367-79.