Intermediate-Conductance Calcium-Activated Potassium Channels
"Intermediate-Conductance Calcium-Activated Potassium Channels" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A major class of calcium-activated potassium channels that were originally discovered in ERYTHROCYTES. They are found primarily in non-excitable CELLS and set up electrical gradients for PASSIVE ION TRANSPORT.
| Descriptor ID |
D051660
|
| MeSH Number(s) |
D12.776.157.530.400.600.150.249 D12.776.543.550.450.750.150.249 D12.776.543.585.400.750.150.249
|
| Concept/Terms |
Intermediate-Conductance Calcium-Activated Potassium Channels- Intermediate-Conductance Calcium-Activated Potassium Channels
- Intermediate Conductance Calcium Activated Potassium Channels
- Potassium Channels, Intermediate-Conductance Calcium-Activated
- Potassium Channels, Intermediate Conductance Calcium Activated
- IK Potassium Channels
- Potassium Channels, IK
|
Below are MeSH descriptors whose meaning is more general than "Intermediate-Conductance Calcium-Activated Potassium Channels".
Below are MeSH descriptors whose meaning is more specific than "Intermediate-Conductance Calcium-Activated Potassium Channels".
This graph shows the total number of publications written about "Intermediate-Conductance Calcium-Activated Potassium Channels" by people in this website by year, and whether "Intermediate-Conductance Calcium-Activated Potassium Channels" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2001 | 0 | 1 | 1 |
| 2003 | 0 | 2 | 2 |
| 2006 | 0 | 1 | 1 |
| 2007 | 0 | 1 | 1 |
| 2010 | 2 | 0 | 2 |
| 2011 | 0 | 1 | 1 |
| 2012 | 1 | 1 | 2 |
| 2013 | 2 | 1 | 3 |
| 2015 | 0 | 1 | 1 |
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Below are the most recent publications written about "Intermediate-Conductance Calcium-Activated Potassium Channels" by people in Profiles.
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The Clinically Tested Gardos Channel Inhibitor Senicapoc Exhibits Antimalarial Activity. Antimicrob Agents Chemother. 2016 01; 60(1):613-6.
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Nucleoside diphosphate kinase B-activated intermediate conductance potassium channels are critical for neointima formation in mouse carotid arteries. Arterioscler Thromb Vasc Biol. 2015 Aug; 35(8):1852-61.
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Prenatal testosterone induces sex-specific dysfunction in endothelium-dependent relaxation pathways in adult male and female rats. Biol Reprod. 2013 Oct; 89(4):97.
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Blocking KCa3.1 channels increases tumor cell killing by a subpopulation of human natural killer lymphocytes. PLoS One. 2013; 8(10):e76740.
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Bradykinin-induced chemotaxis of human gliomas requires the activation of KCa3.1 and ClC-3. J Neurosci. 2013 Jan 23; 33(4):1427-40.
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De novo gain-of-function KCNT1 channel mutations cause malignant migrating partial seizures of infancy. Nat Genet. 2012 Nov; 44(11):1255-9.
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Differential role of IK and BK potassium channels as mediators of intrinsic and extrinsic apoptotic cell death. Am J Physiol Cell Physiol. 2012 Nov 15; 303(10):C1070-8.
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Transient receptor potential canonical type 3 channels facilitate endothelium-derived hyperpolarization-mediated resistance artery vasodilator activity. Cardiovasc Res. 2012 Sep 01; 95(4):439-47.
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Improvements in haemolysis and indicators of erythrocyte survival do not correlate with acute vaso-occlusive crises in patients with sickle cell disease: a phase III randomized, placebo-controlled, double-blind study of the Gardos channel blocker senicapoc (ICA-17043). Br J Haematol. 2011 Apr; 153(1):92-104.
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Functional significance of the intermediate conductance Ca2+-activated K+ channel for the short-term survival of injured erythrocytes. Pflugers Arch. 2010 Nov; 460(6):1029-44.