Potassium Channels, Tandem Pore Domain
"Potassium Channels, Tandem Pore Domain" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Potassium channels that contain two pores in tandem. They are responsible for baseline or leak currents and may be the most numerous of all K channels.
| Descriptor ID |
D024683
|
| MeSH Number(s) |
D12.776.157.530.400.600.850 D12.776.543.550.450.750.850 D12.776.543.585.400.750.850
|
| Concept/Terms |
Potassium Channels, Tandem Pore Domain- Potassium Channels, Tandem Pore Domain
- Potassium Channels, Leak
- Leak Potassium Channels
- Potassium Channels, Baseline
- Baseline Potassium Channels
- K+ Channels, Tandem Pore Domain
- Potassium Channel, Baseline
- Baseline Potassium Channel
- Potassium Channel, Tandem Pore Domain
- Tandem Pore Domain Potassium Channel
- Potassium Channel, Leak
- Leak Potassium Channel
- Potassium Channel, Background
- Background Potassium Channel
- Potassium Channels, Background
- Background Potassium Channels
- Tandem Pore Domain Potassium Channels
|
Below are MeSH descriptors whose meaning is more general than "Potassium Channels, Tandem Pore Domain".
Below are MeSH descriptors whose meaning is more specific than "Potassium Channels, Tandem Pore Domain".
This graph shows the total number of publications written about "Potassium Channels, Tandem Pore Domain" by people in this website by year, and whether "Potassium Channels, Tandem Pore Domain" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2006 | 1 | 0 | 1 |
| 2009 | 2 | 0 | 2 |
| 2010 | 2 | 0 | 2 |
| 2011 | 1 | 0 | 1 |
| 2013 | 1 | 1 | 2 |
| 2014 | 3 | 1 | 4 |
| 2019 | 1 | 0 | 1 |
| 2022 | 1 | 0 | 1 |
| 2024 | 1 | 0 | 1 |
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Below are the most recent publications written about "Potassium Channels, Tandem Pore Domain" by people in Profiles.
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An evolutionarily conserved AnkyrinG-dependent motif clusters axonal K2P K+ channels. J Cell Biol. 2024 Oct 07; 223(10).
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Gain and loss of TASK3 channel function and its regulation by novel variation cause KCNK9 imprinting syndrome. Genome Med. 2022 06 13; 14(1):62.
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Twik-2-/- mouse demonstrates pulmonary vascular heterogeneity in intracellular pathways for vasocontractility. Physiol Rep. 2019 01; 7(1):e13950.
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Inverse remodelling of K2P3.1 K+ channel expression and action potential duration in left ventricular dysfunction and atrial fibrillation: implications for patient-specific antiarrhythmic drug therapy. Eur Heart J. 2017 Jun 07; 38(22):1764-1774.
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Dysregulation of a potassium channel, THIK-1, targeted by caspase-8 accelerates cell shrinkage. Biochim Biophys Acta. 2016 11; 1863(11):2766-2783.
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Response to Letter Regarding Article, "Upregulation of K2P3.1 K+ Current Causes Action Potential Shortening in Patients With Chronic Atrial Fibrillation". Circulation. 2016 Mar 15; 133(11):e440-1.
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Upregulation of K(2P)3.1 K+ Current Causes Action Potential Shortening in Patients With Chronic Atrial Fibrillation. Circulation. 2015 Jul 14; 132(2):82-92.
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Deficiency of the two-pore-domain potassium channel TREK-1 promotes hyperoxia-induced lung injury. Crit Care Med. 2014 Nov; 42(11):e692-701.
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TWIK-2 channel deficiency leads to pulmonary hypertension through a rho-kinase-mediated process. Hypertension. 2014 Dec; 64(6):1260-5.
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Novel approach identifies SNPs in SLC2A10 and KCNK9 with evidence for parent-of-origin effect on body mass index. PLoS Genet. 2014 Jul; 10(7):e1004508.