"Sodium-Calcium Exchanger" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An electrogenic ion exchange protein that maintains a steady level of calcium by removing an amount of calcium equal to that which enters the cells. It is widely distributed in most excitable membranes, including the brain and heart.
| Descriptor ID |
D019831
|
| MeSH Number(s) |
D12.776.157.530.450.162.442 D12.776.543.585.450.162.442
|
| Concept/Terms |
Sodium-Calcium Exchanger- Sodium-Calcium Exchanger
- Exchanger, Sodium-Calcium
- Sodium Calcium Exchanger
- Sodium-Calcium Antiporter
- Antiporter, Sodium-Calcium
- Sodium Calcium Antiporter
- Calcium-Sodium Carrier
- Calcium Sodium Carrier
- Carrier, Calcium-Sodium
- Sodium-Calcium Carrier
- Carrier, Sodium-Calcium
- Sodium Calcium Carrier
- Na(+)-Ca(2+) Exchanger
- Na(+)-Ca(2+) Antiporter
- Calcium-Sodium Exchanger
- Calcium Sodium Exchanger
- Exchanger, Calcium-Sodium
- Ca(2+)-Na(+) Exchanger
- Calcium-Sodium Antiporter
- Antiporter, Calcium-Sodium
- Calcium Sodium Antiporter
- Ca(2+)-Na(+) Antiporter
|
Below are MeSH descriptors whose meaning is more general than "Sodium-Calcium Exchanger".
Below are MeSH descriptors whose meaning is more specific than "Sodium-Calcium Exchanger".
This graph shows the total number of publications written about "Sodium-Calcium Exchanger" by people in this website by year, and whether "Sodium-Calcium Exchanger" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2008 | 0 | 1 | 1 |
| 2010 | 0 | 1 | 1 |
| 2012 | 2 | 0 | 2 |
| 2013 | 0 | 1 | 1 |
| 2014 | 1 | 0 | 1 |
| 2016 | 0 | 1 | 1 |
| 2022 | 1 | 1 | 2 |
| 2023 | 0 | 1 | 1 |
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Below are the most recent publications written about "Sodium-Calcium Exchanger" by people in Profiles.
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Reduction in Junctophilin 2 Expression in Cardiac Nodal Tissue Results in Intracellular Calcium-Driven Increase in Nodal Cell Automaticity. Circ Arrhythm Electrophysiol. 2023 02; 16(2):e010858.
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DMGV Is a Rheostat of T Cell Survival and a Potential Therapeutic for Inflammatory Diseases and Cancers. Front Immunol. 2022; 13:918241.
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Biallelic variants in SLC38A3 encoding a glutamine transporter cause epileptic encephalopathy. Brain. 2022 04 29; 145(3):909-924.
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Intracellular sodium elevation reprograms cardiac metabolism. Nat Commun. 2020 08 28; 11(1):4337.
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Targeting mitochondrial calcium transport in myocardial infarction. Hellenic J Cardiol. 2018 Jul - Aug; 59(4):223-225.
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Cardiac expression of the CREM repressor isoform CREM-Ib?C-X in mice leads to arrhythmogenic alterations in ventricular cardiomyocytes. Basic Res Cardiol. 2016 Mar; 111(2):15.
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Human G109E-inhibitor-1 impairs cardiac function and promotes arrhythmias. J Mol Cell Cardiol. 2015 Dec; 89(Pt B):349-59.
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Reduced junctional Na+/Ca2+-exchanger activity contributes to sarcoplasmic reticulum Ca2+ leak in junctophilin-2-deficient mice. Am J Physiol Heart Circ Physiol. 2014 Nov 01; 307(9):H1317-26.
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Cellular and molecular mechanisms of atrial arrhythmogenesis in patients with paroxysmal atrial fibrillation. Circulation. 2014 Jan 14; 129(2):145-156.
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Cardiac G-protein-coupled receptor kinase 2 ablation induces a novel Ca2+ handling phenotype resistant to adverse alterations and remodeling after myocardial infarction. Circulation. 2012 May 01; 125(17):2108-18.