COUP Transcription Factor I
"COUP Transcription Factor I" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A COUP transcription factor that was originally identified as a homodimer that binds to a direct repeat regulatory element in the chicken albumin promoter. It is a transcription factor that plays an important role in EMBRYONIC DEVELOPMENT of the CENTRAL NERVOUS SYSTEM.
| Descriptor ID |
D051838
|
| MeSH Number(s) |
D12.776.260.643.039.249 D12.776.826.209.039.249
|
| Concept/Terms |
COUP Transcription Factor I- COUP Transcription Factor I
- COUP-TF1 Protein
- COUP TF1 Protein
- Nuclear Receptor Subfamily 2 Group F Member 1
- ErbA-Related Protein-3
- ErbA Related Protein 3
- Nuclear Receptor NR2F1
- Receptor NR2F1, Nuclear
- Chicken Ovalbumin Upstream Promoter-Transcription Factor I
- Chicken Ovalbumin Upstream Promoter Transcription Factor I
- COUP-TFI Protein
- COUP TFI Protein
|
Below are MeSH descriptors whose meaning is more general than "COUP Transcription Factor I".
Below are MeSH descriptors whose meaning is more specific than "COUP Transcription Factor I".
This graph shows the total number of publications written about "COUP Transcription Factor I" by people in this website by year, and whether "COUP Transcription Factor I" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 0 | 3 | 3 |
| 1995 | 0 | 3 | 3 |
| 1996 | 0 | 3 | 3 |
| 1997 | 0 | 3 | 3 |
| 1999 | 0 | 2 | 2 |
| 2000 | 0 | 1 | 1 |
| 2001 | 0 | 1 | 1 |
| 2002 | 0 | 2 | 2 |
| 2004 | 0 | 1 | 1 |
| 2005 | 0 | 1 | 1 |
| 2006 | 1 | 1 | 2 |
| 2007 | 1 | 1 | 2 |
| 2010 | 1 | 1 | 2 |
| 2013 | 1 | 0 | 1 |
| 2014 | 1 | 0 | 1 |
| 2020 | 2 | 0 | 2 |
| 2022 | 0 | 1 | 1 |
| 2023 | 0 | 1 | 1 |
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Below are the most recent publications written about "COUP Transcription Factor I" by people in Profiles.
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Common Neuroimaging Findings in Bosch-Boonstra-Schaaf Optic Atrophy Syndrome. AJNR Am J Neuroradiol. 2023 02; 44(2):212-217.
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Pathophysiological Heterogeneity of the BBSOA Neurodevelopmental Syndrome. Cells. 2022 04 08; 11(8).
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ZEB1-regulated lnc-Nr2f1 promotes the migration and invasion of lung adenocarcinoma cells. Cancer Lett. 2022 05 01; 533:215601.
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Phenotypic expansion of Bosch-Boonstra-Schaaf optic atrophy syndrome and further evidence for genotype-phenotype correlations. Am J Med Genet A. 2020 06; 182(6):1426-1437.
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Nr2f1 heterozygous knockout mice recapitulate neurological phenotypes of Bosch-Boonstra-Schaaf optic atrophy syndrome and show impaired hippocampal synaptic plasticity. Hum Mol Genet. 2020 03 27; 29(5):705-715.
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Mitochondrial involvement in a Bosch-Boonstra-Schaaf optic atrophy syndrome patient with a novel de novo NR2F1 gene mutation. J Hum Genet. 2018 Apr; 63(4):525-528.
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The expanding clinical phenotype of Bosch-Boonstra-Schaaf optic atrophy syndrome: 20 new cases and possible genotype-phenotype correlations. Genet Med. 2016 11; 18(11):1143-1150.
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NR2F1 mutations cause optic atrophy with intellectual disability. Am J Hum Genet. 2014 Feb 06; 94(2):303-9.
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A coregulatory network of NR2F1 and microRNA-140. PLoS One. 2013; 8(12):e83358.
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A gene signature identified using a mouse model of androgen receptor-dependent prostate cancer predicts biochemical relapse in human disease. Int J Cancer. 2012 Aug 01; 131(3):662-72.