"Calcium Channels, N-Type" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
CALCIUM CHANNELS that are concentrated in neural tissue. Omega toxins inhibit the actions of these channels by altering their voltage dependence.
| Descriptor ID |
D020864
|
| MeSH Number(s) |
D12.776.157.530.400.150.585 D12.776.543.550.450.150.585 D12.776.543.585.400.150.585
|
| Concept/Terms |
Calcium Channels, N-Type- Calcium Channels, N-Type
- Calcium Channels, N Type
- Channels, N-Type Calcium
- N-Type VDCC
- N Type VDCC
- VDCC, N-Type
- N-Type Calcium Channel
- Calcium Channel, N-Type
- Channel, N-Type Calcium
- N Type Calcium Channel
- N-Type Voltage-Dependent Calcium Channels
- N Type Voltage Dependent Calcium Channels
- N-Type Calcium Channels
- N Type Calcium Channels
- Neural-Type Calcium Channels
- Calcium Channels, Neural-Type
- Channels, Neural-Type Calcium
- Neural Type Calcium Channels
|
Below are MeSH descriptors whose meaning is more general than "Calcium Channels, N-Type".
Below are MeSH descriptors whose meaning is more specific than "Calcium Channels, N-Type".
This graph shows the total number of publications written about "Calcium Channels, N-Type" by people in this website by year, and whether "Calcium Channels, N-Type" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1997 | 1 | 0 | 1 |
| 1999 | 1 | 0 | 1 |
| 2000 | 1 | 0 | 1 |
| 2001 | 0 | 2 | 2 |
| 2002 | 0 | 1 | 1 |
| 2007 | 0 | 2 | 2 |
| 2008 | 1 | 0 | 1 |
| 2013 | 1 | 0 | 1 |
| 2014 | 1 | 0 | 1 |
| 2015 | 1 | 1 | 2 |
| 2016 | 1 | 0 | 1 |
| 2017 | 0 | 1 | 1 |
| 2020 | 1 | 0 | 1 |
| 2022 | 1 | 1 | 2 |
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Below are the most recent publications written about "Calcium Channels, N-Type" by people in Profiles.
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Biallelic CACNA2D1 loss-of-function variants cause early-onset developmental epileptic encephalopathy. Brain. 2022 08 27; 145(8):2721-2729.
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Complex effects on CaV2.1 channel gating caused by a CACNA1A variant associated with a severe neurodevelopmental disorder. Sci Rep. 2022 06 02; 12(1):9186.
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Adult loss of Cacna1a in mice recapitulates childhood absence epilepsy by distinct thalamic bursting mechanisms. Brain. 2020 01 01; 143(1):161-174.
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Persistent aberrant cortical phase-amplitude coupling following seizure treatment in absence epilepsy models. J Physiol. 2017 12 01; 595(23):7249-7260.
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Isolated P/Q Calcium Channel Deletion in Layer VI Corticothalamic Neurons Generates Absence Epilepsy. J Neurosci. 2016 Jan 13; 36(2):405-18.
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Shift in interictal relative gamma power as a novel biomarker for drug response in two mouse models of absence epilepsy. Epilepsia. 2016 Jan; 57(1):79-88.
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Molecular Basis of Regulating High Voltage-Activated Calcium Channels by S-Nitrosylation. J Biol Chem. 2015 Dec 18; 290(51):30616-23.
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A voltage-gated calcium channel regulates lysosomal fusion with endosomes and autophagosomes and is required for neuronal homeostasis. PLoS Biol. 2015 Mar; 13(3):e1002103.
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Potentiation of high voltage-activated calcium channels by 4-aminopyridine depends on subunit composition. Mol Pharmacol. 2014 Dec; 86(6):760-72.
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Compromised maturation of GABAergic inhibition underlies abnormal network activity in the hippocampus of epileptic Ca2+ channel mutant mice, tottering. Pflugers Arch. 2015 Apr; 467(4):737-52.