M Phase Cell Cycle Checkpoints
"M Phase Cell Cycle Checkpoints" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The cellular signaling system that halts the progression of cells through MITOSIS or MEIOSIS if a defect that will affect CHROMOSOME SEGREGATION is detected.
| Descriptor ID |
D059566
|
| MeSH Number(s) |
G04.144.109.750 G04.144.220.220.781.337 G05.113.220.781.338
|
| Concept/Terms |
M Phase Cell Cycle Checkpoints- M Phase Cell Cycle Checkpoints
- M Phase Cell Cycle Checkpoint
- M Phase Checkpoint
- Checkpoint, M Phase
- Checkpoints, M Phase
- M Phase Checkpoints
- Phase Checkpoint, M
Spindle Assembly Checkpoint- Spindle Assembly Checkpoint
- Assembly Checkpoint, Spindle
- Assembly Checkpoints, Spindle
- Checkpoint, Spindle Assembly
- Checkpoints, Spindle Assembly
- Spindle Assembly Checkpoints
Mitotic Checkpoints- Mitotic Checkpoints
- Checkpoint, Mitotic
- Checkpoints, Mitotic
- Mitotic Checkpoint
Mitotic Spindle Checkpoints- Mitotic Spindle Checkpoints
- Checkpoint, Mitotic Spindle
- Checkpoints, Mitotic Spindle
- Mitotic Spindle Checkpoint
- Spindle Checkpoint, Mitotic
- Spindle Checkpoints, Mitotic
|
Below are MeSH descriptors whose meaning is more general than "M Phase Cell Cycle Checkpoints".
Below are MeSH descriptors whose meaning is more specific than "M Phase Cell Cycle Checkpoints".
This graph shows the total number of publications written about "M Phase Cell Cycle Checkpoints" by people in this website by year, and whether "M Phase Cell Cycle Checkpoints" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2012 | 1 | 0 | 1 |
| 2013 | 0 | 2 | 2 |
| 2016 | 1 | 0 | 1 |
| 2018 | 0 | 1 | 1 |
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Below are the most recent publications written about "M Phase Cell Cycle Checkpoints" by people in Profiles.
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A YAP/FOXM1 axis mediates EMT-associated EGFR inhibitor resistance and increased expression of spindle assembly checkpoint components. Sci Transl Med. 2020 09 02; 12(559).
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Dual compartmental targeting of cell cycle and angiogenic kinases in colorectal cancer models. Anticancer Drugs. 2018 10; 29(9):827-838.
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The BUB3-BUB1 Complex Promotes Telomere DNA Replication. Mol Cell. 2018 05 03; 70(3):395-407.e4.
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Glioblastoma stem cells exploit the av?8 integrin-TGF?1 signaling axis to drive tumor initiation and progression. Oncogene. 2017 11 23; 36(47):6568-6580.
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Histone H3K4 methylation regulates deactivation of the spindle assembly checkpoint through direct binding of Mad2. Genes Dev. 2016 05 15; 30(10):1187-97.
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Inhibition of P-TEFb by DRB suppresses SIRT1/CK2a pathway and enhances radiosensitivity of human cancer cells. Anticancer Res. 2014 Dec; 34(12):6981-9.
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Proteasome inhibitors evoke latent tumor suppression programs in pro-B MLL leukemias through MLL-AF4. Cancer Cell. 2014 Apr 14; 25(4):530-42.
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Expression profiling stratifies mesothelioma tumors and signifies deregulation of spindle checkpoint pathway and microtubule network with therapeutic implications. Ann Oncol. 2014 Jun; 25(6):1184-92.
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Benzopyran derivative CDRI-85/287 induces G2-M arrest in estrogen receptor-positive breast cancer cells via modulation of estrogen receptors a- and ?-mediated signaling, in parallel to EGFR signaling and suppresses the growth of tumor xenograft. Steroids. 2013 Nov; 78(11):1071-86.
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The tumor suppressor SirT2 regulates cell cycle progression and genome stability by modulating the mitotic deposition of H4K20 methylation. Genes Dev. 2013 Mar 15; 27(6):639-53.