"Elapid Venoms" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Venoms from snakes of the family Elapidae, including cobras, kraits, mambas, coral, tiger, and Australian snakes. The venoms contain polypeptide toxins of various kinds, cytolytic, hemolytic, and neurotoxic factors, but fewer enzymes than viper or crotalid venoms. Many of the toxins have been characterized.
| Descriptor ID |
D004546
|
| MeSH Number(s) |
D20.888.850.325 D23.946.833.850.325
|
| Concept/Terms |
Elapid Venoms- Elapid Venoms
- Venoms, Elapid
- Elapid Venom
- Venom, Elapid
Cobra Venoms- Cobra Venoms
- Venoms, Cobra
- Naja Venoms
- Venoms, Naja
- Naja Venom
- Venom, Naja
- Cobra Venom
- Venom, Cobra
Hydrophid Venoms- Hydrophid Venoms
- Venoms, Hydrophid
- Sea Snake Venoms
- Venoms, Sea Snake
- Sea Snake Venom
- Venom, Sea Snake
- Hydrophid Venom
- Venom, Hydrophid
King Cobra Venom- King Cobra Venom
- Venom, King Cobra
- Ophiophagus hannah Venom
- Venom, Ophiophagus hannah
|
Below are MeSH descriptors whose meaning is more general than "Elapid Venoms".
Below are MeSH descriptors whose meaning is more specific than "Elapid Venoms".
This graph shows the total number of publications written about "Elapid Venoms" by people in this website by year, and whether "Elapid Venoms" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1996 | 1 | 0 | 1 |
| 2000 | 0 | 1 | 1 |
| 2004 | 0 | 2 | 2 |
| 2006 | 0 | 1 | 1 |
| 2011 | 0 | 1 | 1 |
| 2022 | 0 | 1 | 1 |
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Below are the most recent publications written about "Elapid Venoms" by people in Profiles.
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Successful management of monocled cobra (Naja kaouthia) envenomation with neuro polyvalent antivenom. Clin Toxicol (Phila). 2022 09; 60(9):1074-1075.
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Alternative splicing dysregulation secondary to skeletal muscle regeneration. Ann Neurol. 2011 Apr; 69(4):681-90.
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Human macrophages rescue myoblasts and myotubes from apoptosis through a set of adhesion molecular systems. J Cell Sci. 2006 Jun 15; 119(Pt 12):2497-507.
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Mu opioid receptor activation inhibits GABAergic inputs to basolateral amygdala neurons through Kv1.1/1.2 channels. J Neurophysiol. 2006 Apr; 95(4):2032-41.
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Alterations in the natriuretic hormone system related to cardiopulmonary bypass in infants with congestive heart failure. Pediatr Cardiol. 2004 Jul-Aug; 25(4):347-53.
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[Ion channels and demyelination: basis of a treatment of experimental autoimmune encephalomyelitis (EAE) by potassium channel blockers]. Rev Neurol (Paris). 2004 May; 160(5 Pt 2):S16-27.
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Biochemical interactions of the neuronal pentraxins. Neuronal pentraxin (NP) receptor binds to taipoxin and taipoxin-associated calcium-binding protein 49 via NP1 and NP2. J Biol Chem. 2000 Jun 09; 275(23):17786-92.
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Complement activation following oxidative stress. Mol Immunol. 1999 Sep-Oct; 36(13-14):941-8.
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k-Opioid receptor activation of a dendrotoxin-sensitive potassium channel mediates presynaptic inhibition of mossy fiber neurotransmitter release. Mol Pharmacol. 1996 Jul; 50(1):80-5.
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Oleic-acid-induced lung injury in the rat. Failure of indomethacin treatment or complement depletion to ablate lung injury. Am J Pathol. 1981 Jun; 103(3):376-83.