Miniature Postsynaptic Potentials
"Miniature Postsynaptic Potentials" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Postsynaptic potentials generated from a release of neurotransmitters from a presynaptic nerve terminal in the absence of an ACTION POTENTIAL. They may be m.e.p.p.s (miniature EXCITATORY POSTSYNAPTIC POTENTIALS) or m.i.p.p.s (miniature INHIBITORY POSTSYNAPTIC POTENTIALS).
| Descriptor ID |
D055369
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| MeSH Number(s) |
G04.580.887.500 G07.265.675.887.500 G07.265.880.750.560 G11.561.570.918.500 G11.561.830.750.560
|
| Concept/Terms |
Miniature Postsynaptic Potentials- Miniature Postsynaptic Potentials
- Miniature Postsynaptic Potential
- Postsynaptic Potential, Miniature
- Postsynaptic Potentials, Miniature
- Potential, Miniature Postsynaptic
- Potentials, Miniature Postsynaptic
Spontaneous Postsynaptic Potentials- Spontaneous Postsynaptic Potentials
- Postsynaptic Potential, Spontaneous
- Postsynaptic Potentials, Spontaneous
- Potential, Spontaneous Postsynaptic
- Potentials, Spontaneous Postsynaptic
- Spontaneous Postsynaptic Potential
|
Below are MeSH descriptors whose meaning is more general than "Miniature Postsynaptic Potentials".
Below are MeSH descriptors whose meaning is more specific than "Miniature Postsynaptic Potentials".
This graph shows the total number of publications written about "Miniature Postsynaptic Potentials" by people in this website by year, and whether "Miniature Postsynaptic Potentials" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2011 | 0 | 1 | 1 |
| 2024 | 1 | 0 | 1 |
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Below are the most recent publications written about "Miniature Postsynaptic Potentials" by people in Profiles.
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Giant miniature endplate potentials at vertebrate neuromuscular junctions: A review. Eur J Neurosci. 2024 Dec; 60(12):7183-7194.
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Nitric oxide stimulates glutamatergic synaptic inputs to baroreceptor neurons through potentiation of Cav2.2-mediated Ca(2+) currents. Neurosci Lett. 2014 May 01; 567:57-62.
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Casein kinase II inhibition reverses pain hypersensitivity and potentiated spinal N-methyl-D-aspartate receptor activity caused by calcineurin inhibitor. J Pharmacol Exp Ther. 2014 May; 349(2):239-47.
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Calcineurin inhibitor induces pain hypersensitivity by potentiating pre- and postsynaptic NMDA receptor activity in spinal cords. J Physiol. 2014 Jan 01; 592(1):215-27.
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Disruption of the non-canonical Wnt gene PRICKLE2 leads to autism-like behaviors with evidence for hippocampal synaptic dysfunction. Mol Psychiatry. 2013 Oct; 18(10):1077-89.
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Interplay between VGLUT isoforms and endophilin A1 regulates neurotransmitter release and short-term plasticity. Neuron. 2011 Mar 24; 69(6):1147-59.