Large-Conductance Calcium-Activated Potassium Channel beta Subunits
"Large-Conductance Calcium-Activated Potassium Channel beta Subunits" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The regulatory subunits of large-conductance calcium-activated potassium channels.
| Descriptor ID |
D051038
|
| MeSH Number(s) |
D12.776.157.530.400.600.150.500.750 D12.776.543.550.450.750.150.500.750 D12.776.543.585.400.750.150.500.500
|
| Concept/Terms |
Large-Conductance Calcium-Activated Potassium Channel beta Subunits- Large-Conductance Calcium-Activated Potassium Channel beta Subunits
- Large Conductance Calcium Activated Potassium Channel beta Subunits
- Large-Conductance Calcium-Activated Potassium Channels, beta Subunits
- Large Conductance Calcium Activated Potassium Channels, beta Subunits
- MaxiK Channel beta Subunits
|
Below are MeSH descriptors whose meaning is more general than "Large-Conductance Calcium-Activated Potassium Channel beta Subunits".
Below are MeSH descriptors whose meaning is more specific than "Large-Conductance Calcium-Activated Potassium Channel beta Subunits".
This graph shows the total number of publications written about "Large-Conductance Calcium-Activated Potassium Channel beta Subunits" by people in this website by year, and whether "Large-Conductance Calcium-Activated Potassium Channel beta Subunits" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2021 | 1 | 0 | 1 |
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Below are the most recent publications written about "Large-Conductance Calcium-Activated Potassium Channel beta Subunits" by people in Profiles.
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Analysis of genes (TMEM106B, GRN, ABCC9, KCNMB2, and APOE) implicated in risk for LATE-NC and hippocampal sclerosis provides pathogenetic insights: a retrospective genetic association study. Acta Neuropathol Commun. 2021 09 15; 9(1):152.
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Beta1-subunits increase surface expression of a large-conductance Ca2+-activated K+ channel isoform. J Neurophysiol. 2007 May; 97(5):3508-16.