"G1 Phase" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The period of the CELL CYCLE preceding DNA REPLICATION in S PHASE. Subphases of G1 include "competence" (to respond to growth factors), G1a (entry into G1), G1b (progression), and G1c (assembly). Progression through the G1 subphases is effected by limiting growth factors, nutrients, or inhibitors.
Descriptor ID |
D016193
|
MeSH Number(s) |
G04.144.500.320
|
Concept/Terms |
G1 Phase- G1 Phase
- G1 Phases
- Phase, G1
- Phases, G1
- First Gap Phase
- First Gap Phases
- Gap Phase, First
- Gap Phases, First
- Phase, First Gap
- Phases, First Gap
- Gap Phase 1
- Phase 1, Gap
G1b Phase- G1b Phase
- G1b Phases
- Phase, G1b
- Phases, G1b
G1a Phase- G1a Phase
- G1a Phases
- Phase, G1a
- Phases, G1a
|
Below are MeSH descriptors whose meaning is more general than "G1 Phase".
Below are MeSH descriptors whose meaning is more specific than "G1 Phase".
This graph shows the total number of publications written about "G1 Phase" by people in this website by year, and whether "G1 Phase" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
Year | Major Topic | Minor Topic | Total |
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1994 | 3 | 5 | 8 |
1995 | 1 | 4 | 5 |
1996 | 3 | 6 | 9 |
1997 | 1 | 4 | 5 |
1998 | 3 | 4 | 7 |
1999 | 5 | 10 | 15 |
2000 | 3 | 8 | 11 |
2001 | 5 | 10 | 15 |
2002 | 2 | 7 | 9 |
2003 | 2 | 14 | 16 |
2004 | 3 | 16 | 19 |
2005 | 2 | 8 | 10 |
2006 | 1 | 11 | 12 |
2007 | 1 | 9 | 10 |
2008 | 0 | 4 | 4 |
2009 | 1 | 13 | 14 |
2010 | 1 | 6 | 7 |
2011 | 2 | 5 | 7 |
2012 | 0 | 1 | 1 |
2013 | 2 | 3 | 5 |
2015 | 1 | 2 | 3 |
2016 | 0 | 1 | 1 |
2018 | 0 | 2 | 2 |
2020 | 1 | 0 | 1 |
2021 | 0 | 1 | 1 |
2024 | 1 | 0 | 1 |
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Below are the most recent publications written about "G1 Phase" by people in Profiles.
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Reactivation of the G1 enhancer landscape underlies core circuitry addiction to SWI/SNF. Nucleic Acids Res. 2024 Jan 11; 52(1):4-21.
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A novel role of ADGRF1 (GPR110) in promoting cellular quiescence and chemoresistance in human epidermal growth factor receptor 2-positive breast cancer. FASEB J. 2021 07; 35(7):e21719.
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Specific, reversible G1 arrest by UCN-01 in vivo provides cytostatic protection of normal cells against cytotoxic chemotherapy in breast cancer. Br J Cancer. 2020 03; 122(6):812-822.
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Minichromosome Maintenance Proteins Cooperate with LANA during the G1/S Phase of the Cell Cycle To Support Viral DNA Replication. J Virol. 2019 04 01; 93(7).
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Loss of p53 expression in cancer cells alters cell cycle response after inhibition of exportin-1 but does not prevent cell death. Cell Cycle. 2018; 17(11):1329-1344.
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Centromere transcription allows CENP-A to transit from chromatin association to stable incorporation. J Cell Biol. 2018 06 04; 217(6):1957-1972.
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Tideglusib induces apoptosis in human neuroblastoma IMR32 cells, provoking sub-G0/G1 accumulation and ROS generation. Environ Toxicol Pharmacol. 2016 Sep; 46:194-205.
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Estrogen receptor alpha is cell cycle-regulated and regulates the cell cycle in a ligand-dependent fashion. Cell Cycle. 2016 06 17; 15(12):1579-90.
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CDK5 Regulates Paclitaxel Sensitivity in Ovarian Cancer Cells by Modulating AKT Activation, p21Cip1- and p27Kip1-Mediated G1 Cell Cycle Arrest and Apoptosis. PLoS One. 2015; 10(7):e0131833.
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Role of miR-145 in human laryngeal squamous cell carcinoma. Head Neck. 2016 Feb; 38(2):260-6.