"Mice, Inbred NOD" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A strain of non-obese diabetic mice developed in Japan that has been widely studied as a model for T-cell-dependent autoimmune insulin-dependent diabetes mellitus in which insulitis is a major histopathologic feature, and in which genetic susceptibility is strongly MHC-linked.
| Descriptor ID |
D016688
|
| MeSH Number(s) |
B01.050.050.199.520.520.565 B01.050.150.900.649.313.992.635.505.500.400.565
|
| Concept/Terms |
Mice, Inbred NOD- Mice, Inbred NOD
- Inbred NOD Mice
- NOD Mice, Inbred
- Mouse, NOD
- NOD Mouse
- Nonobese Diabetic Mice
- Diabetic Mice, Nonobese
- Mice, Nonobese Diabetic
- Non-Obese Diabetic Mouse
- Diabetic Mouse, Non-Obese
- Mouse, Non-Obese Diabetic
- Non Obese Diabetic Mouse
- Non-Obese Diabetic Mice
- Diabetic Mice, Non-Obese
- Mice, Non-Obese Diabetic
- Non Obese Diabetic Mice
- Mouse, Inbred NOD
- Inbred NOD Mouse
- NOD Mouse, Inbred
- Mice, NOD
- NOD Mice
- Nonobese Diabetic Mouse
- Diabetic Mouse, Nonobese
- Mouse, Nonobese Diabetic
|
Below are MeSH descriptors whose meaning is more general than "Mice, Inbred NOD".
Below are MeSH descriptors whose meaning is more specific than "Mice, Inbred NOD".
This graph shows the total number of publications written about "Mice, Inbred NOD" by people in this website by year, and whether "Mice, Inbred NOD" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1997 | 1 | 3 | 4 |
| 1999 | 0 | 1 | 1 |
| 2000 | 0 | 1 | 1 |
| 2001 | 0 | 2 | 2 |
| 2002 | 0 | 5 | 5 |
| 2003 | 0 | 2 | 2 |
| 2004 | 0 | 5 | 5 |
| 2005 | 0 | 6 | 6 |
| 2007 | 0 | 9 | 9 |
| 2008 | 0 | 8 | 8 |
| 2009 | 0 | 15 | 15 |
| 2010 | 0 | 16 | 16 |
| 2011 | 0 | 19 | 19 |
| 2012 | 0 | 33 | 33 |
| 2013 | 0 | 27 | 27 |
| 2014 | 0 | 39 | 39 |
| 2015 | 0 | 27 | 27 |
| 2016 | 0 | 38 | 38 |
| 2017 | 0 | 23 | 23 |
| 2018 | 0 | 43 | 43 |
| 2019 | 0 | 38 | 38 |
| 2020 | 0 | 24 | 24 |
| 2021 | 0 | 39 | 39 |
| 2022 | 0 | 3 | 3 |
| 2023 | 0 | 6 | 6 |
| 2024 | 0 | 2 | 2 |
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click here.
Below are the most recent publications written about "Mice, Inbred NOD" by people in Profiles.
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Hu8F4-CAR T cells with mutated Fc spacer segment improve target specificity and mediate anti-leukemia activity in vivo. Cytotherapy. 2024 Nov; 26(11):1331-1340.
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Integration of ?-deficient CARs into the CD3? gene conveys potent cytotoxicity in T and NK cells. Blood. 2024 06 20; 143(25):2599-2611.
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Persistent organic pollutants promote aggressiveness in prostate cancer. Oncogene. 2023 09; 42(38):2854-2867.
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Targeting GBM with an Oncolytic Picornavirus SVV-001 alone and in combination with fractionated Radiation in a Novel Panel of Orthotopic PDX models. J Transl Med. 2023 07 06; 21(1):444.
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Interaction between tumor cell TNFR2 and monocyte membrane-bound TNF-a triggers tumorigenic inflammation in neuroblastoma. J Immunother Cancer. 2023 03; 11(3).
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Lacrimal Gland Epithelial Cells Shape Immune Responses through the Modulation of Inflammasomes and Lipid Metabolism. Int J Mol Sci. 2023 Feb 21; 24(5).
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AGA induces sub-G1 cell cycle arrest and apoptosis in human colon cancer cells through p53-independent/p53-dependent pathway. BMC Cancer. 2023 Jan 02; 23(1):1.
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Anti-Colorectal Cancer Effects of Fucoidan Complex-Based Functional Beverage Through Retarding Proliferation, Cell Cycle and Epithelial-Mesenchymal Transition Signaling Pathways. Integr Cancer Ther. 2023 Jan-Dec; 22:15347354231213613.
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Spatial transcriptomics of the lacrimal gland features macrophage activity and epithelium metabolism as key alterations during chronic inflammation. Front Immunol. 2022; 13:1011125.
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Co-clinical Modeling of the Activity of the BET Inhibitor Mivebresib (ABBV-075) in AML. In Vivo. 2022 Jul-Aug; 36(4):1615-1627.