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CREIGHTON, CHAD
One or more keywords matched the following items that are connected to CREIGHTON, CHAD
Item TypeName
Academic Article ZEB1 drives prometastatic actin cytoskeletal remodeling by downregulating miR-34a expression.
Concept Zinc Fingers
Academic Article ZEB1 sensitizes lung adenocarcinoma to metastasis suppression by PI3K antagonism.
Academic Article KAP1 promotes proliferation and metastatic progression of breast cancer cells.
Academic Article The miR-200 family and the miR-183~96~182 cluster target Foxf2 to inhibit invasion and metastasis in lung cancers.
Academic Article The microRNA-200/Zeb1 axis regulates ECM-dependent ?1-integrin/FAK signaling, cancer cell invasion and metastasis through CRKL.
Academic Article A genetic cell context-dependent role for ZEB1 in lung cancer.
Academic Article ZEB1 induces LOXL2-mediated collagen stabilization and deposition in the extracellular matrix to drive lung cancer invasion and metastasis.
Academic Article Epithelial-to-mesenchymal transition drives a pro-metastatic Golgi compaction process through scaffolding protein PAQR11.
Academic Article The epithelial-to-mesenchymal transition activator ZEB1 initiates a prometastatic competing endogenous RNA network.
Concept Zinc Finger E-box-Binding Homeobox 1
Concept Zinc Finger E-box Binding Homeobox 2
Academic Article ZEB1/NuRD complex suppresses TBC1D2b to stimulate E-cadherin internalization and promote metastasis in lung cancer.
Academic Article Functional Hierarchy and Cooperation of EMT Master Transcription Factors in Breast Cancer Metastasis.
Academic Article Contextual cues from cancer cells govern cancer-associated fibroblast heterogeneity.
Academic Article The EMT activator ZEB1 accelerates endosomal trafficking to establish a polarity axis in lung adenocarcinoma cells.
Academic Article Transcriptional control of a collagen deposition and adhesion process that promotes lung adenocarcinoma growth and metastasis.
Academic Article EMT-activated secretory and endocytic vesicular trafficking programs underlie a vulnerability to PI4K2A antagonism in lung cancer.
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