N-Terminal Acetyltransferase E
"N-Terminal Acetyltransferase E" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An N-terminal acetyltransferase subtype that consists of the Naa50p catalytic subunit, and the Naa10p and Naa15p auxiliary subunits. It has specificity for the N-terminal METHIONINE of peptides where the next amino acid in the chain is hydrophobic.
| Descriptor ID |
D063213
|
| MeSH Number(s) |
D08.811.913.050.134.423.500
|
| Concept/Terms |
N-Terminal Acetyltransferase E- N-Terminal Acetyltransferase E
- Acetyltransferase E, N-Terminal
- N Terminal Acetyltransferase E
- NatE N-Terminal Acetyltransferase
- Acetyltransferase, NatE N-Terminal
- N-Terminal Acetyltransferase, NatE
- NatE N Terminal Acetyltransferase
- N-Terminal Acetyltransferase Type E
- N Terminal Acetyltransferase Type E
|
Below are MeSH descriptors whose meaning is more general than "N-Terminal Acetyltransferase E".
Below are MeSH descriptors whose meaning is more specific than "N-Terminal Acetyltransferase E".
This graph shows the total number of publications written about "N-Terminal Acetyltransferase E" by people in this website by year, and whether "N-Terminal Acetyltransferase E" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2018 | 2 | 0 | 2 |
| 2019 | 1 | 0 | 1 |
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Below are the most recent publications written about "N-Terminal Acetyltransferase E" by people in Profiles.
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Phenotypic and biochemical analysis of an international cohort of individuals with variants in NAA10 and NAA15. Hum Mol Genet. 2019 09 01; 28(17):2900-2919.
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A novel NAA10 variant with impaired acetyltransferase activity causes developmental delay, intellectual disability, and hypertrophic cardiomyopathy. Eur J Hum Genet. 2018 09; 26(9):1294-1305.
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Truncating Variants in NAA15 Are Associated with Variable Levels of Intellectual Disability, Autism Spectrum Disorder, and Congenital Anomalies. Am J Hum Genet. 2018 05 03; 102(5):985-994.
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Phosphoglycerate Kinase 1 Phosphorylates Beclin1 to Induce Autophagy. Mol Cell. 2017 Mar 02; 65(5):917-931.e6.
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Using VAAST to identify an X-linked disorder resulting in lethality in male infants due to N-terminal acetyltransferase deficiency. Am J Hum Genet. 2011 Jul 15; 89(1):28-43.
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ARD1 stabilization of TSC2 suppresses tumorigenesis through the mTOR signaling pathway. Sci Signal. 2010 Feb 09; 3(108):ra9.
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Phosphorylation of ARD1 by IKKbeta contributes to its destabilization and degradation. Biochem Biophys Res Commun. 2009 Nov 06; 389(1):156-61.