"DNA Ligase ATP" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
ATP-dependent cellular enzyme which catalyzes DNA replication, repair and recombination through formation of internucleotide ester bonds between phosphate and deoxyribose moieties. Vertebrate cells encode three well-characterized DNA ligases, DNA ligase I, III and IV, all of which are related in structure and sequence. DNA ligases either require ATP or NAD. However, archaebacterial, viral, and some eubacterial DNA ligases are ATP-dependent.
| Descriptor ID |
D000072481
|
| MeSH Number(s) |
D08.811.074.500.500 D08.811.464.754.600.500
|
| Concept/Terms |
DNA Ligase ATP- DNA Ligase ATP
- ATP, DNA Ligase
- Ligase ATP, DNA
- Polydeoxyribonucleotide Synthase ATP
- ATP, Polydeoxyribonucleotide Synthase
- Synthase ATP, Polydeoxyribonucleotide
- DNA Ligases, ATP-Dependent
- ATP-Dependent DNA Ligases
- DNA Ligases, ATP Dependent
- Ligases, ATP-Dependent DNA
DNA Ligase IIIalpha- DNA Ligase IIIalpha
- IIIalpha, DNA Ligase
- Ligase IIIalpha, DNA
- LIGIIIalpha Protein
DNA Ligase III- DNA Ligase III
- Ligase III, DNA
- DNA Ligase II
- Ligase II, DNA
|
Below are MeSH descriptors whose meaning is more general than "DNA Ligase ATP".
Below are MeSH descriptors whose meaning is more specific than "DNA Ligase ATP".
This graph shows the total number of publications written about "DNA Ligase ATP" by people in this website by year, and whether "DNA Ligase ATP" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2002 | 0 | 1 | 1 |
| 2004 | 0 | 1 | 1 |
| 2005 | 0 | 1 | 1 |
| 2008 | 0 | 1 | 1 |
| 2009 | 0 | 1 | 1 |
| 2010 | 0 | 1 | 1 |
| 2011 | 0 | 1 | 1 |
| 2015 | 0 | 1 | 1 |
| 2019 | 1 | 0 | 1 |
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Below are the most recent publications written about "DNA Ligase ATP" by people in Profiles.
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Inhibition of base excision repair by natamycin suppresses prostate cancer cell proliferation. Biochimie. 2020 Jan; 168:241-250.
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Adapting capillary gel electrophoresis as a sensitive, high-throughput method to accelerate characterization of nucleic acid metabolic enzymes. Nucleic Acids Res. 2016 Jan 29; 44(2):e15.
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Ku70 functions in addition to nonhomologous end joining in pancreatic ?-cells: a connection to ?-catenin regulation. Diabetes. 2013 Jul; 62(7):2429-38.
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Genetic determinants for promoter hypermethylation in the lungs of smokers: a candidate gene-based study. Cancer Res. 2012 Feb 01; 72(3):707-15.
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Polymorphisms of LIG4, BTBD2, HMGA2, and RTEL1 genes involved in the double-strand break repair pathway predict glioblastoma survival. J Clin Oncol. 2010 May 10; 28(14):2467-74.
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Absence of p53-dependent apoptosis combined with nonhomologous end-joining deficiency leads to a severe diabetic phenotype in mice. Diabetes. 2010 Jan; 59(1):135-42.
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Association and interactions between DNA repair gene polymorphisms and adult glioma. Cancer Epidemiol Biomarkers Prev. 2009 Jan; 18(1):204-14.
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Polymorphisms of LIG4 and XRCC4 involved in the NHEJ pathway interact to modify risk of glioma. Hum Mutat. 2008 Mar; 29(3):381-9.
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DNA damage-induced cellular senescence is sufficient to suppress tumorigenesis: a mouse model. J Exp Med. 2007 Jun 11; 204(6):1453-61.
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Early embryonic lethality due to targeted inactivation of DNA ligase III. Mol Cell Biol. 2006 May; 26(10):3935-41.