"Job Syndrome" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Primary immunodeficiency syndrome characterized by recurrent infections and hyperimmunoglobulinemia E. Most cases are sporadic. Of the rare familial forms, the dominantly inherited subtype has additional connective tissue, dental and skeletal involvement that the recessive type does not share.
| Descriptor ID |
D007589
|
| MeSH Number(s) |
C15.378.553.774.600 C20.673.774.600
|
| Concept/Terms |
Job Syndrome- Job Syndrome
- Job Syndromes
- Syndrome, Job
- Syndromes, Job
- Hyper-IgE Syndrome
- Hyper IgE Syndrome
- Hyper-IgE Syndromes
- Syndrome, Hyper-IgE
- Hyperimmunoglobulin E, Recurrent Infection Syndrome
- HIE Syndrome
- HIE Syndromes
- Syndrome, HIE
- Syndromes, HIE
- Hyperimmunoglobulinemia E Syndrome
- Hyperimmunoglobulinemia E Syndromes
- Syndrome, Hyperimmunoglobulinemia E
- Syndromes, Hyperimmunoglobulinemia E
- Buckley Syndrome
- Buckley Syndromes
- Syndrome, Buckley
- Syndromes, Buckley
- Job-Buckley Syndrome
- Job Buckley Syndrome
- Job-Buckley Syndromes
- Syndrome, Job-Buckley
- Syndromes, Job-Buckley
- Job's Syndrome
- Jobs Syndrome
- Syndrome, Job's
- Hyperimmunoglobulin E-Recurrent Infection Syndrome
- Hyperimmunoglobulin E Recurrent Infection Syndrome
Hyper-Immunoglobulin E Syndrome, Autosomal Recessive- Hyper-Immunoglobulin E Syndrome, Autosomal Recessive
- Hyper Immunoglobulin E Syndrome, Autosomal Recessive
- Hyper-IgE Recurrent Infection Syndrome, Autosomal Recessive
- Hyper IgE Recurrent Infection Syndrome, Autosomal Recessive
- Hyper-IgE Syndrome, Autosomal Recessive
- Hyper IgE Syndrome, Autosomal Recessive
- HIES, Autosomal Recessive
- Autosomal Recessive HIES
- Autosomal Recessive HIESs
- HIESs, Autosomal Recessive
Hyper-Immunoglobulin E Syndrome, Autosomal Dominant- Hyper-Immunoglobulin E Syndrome, Autosomal Dominant
- Hyper Immunoglobulin E Syndrome, Autosomal Dominant
- Hyper-IgE Syndrome, Autosomal Dominant
- Hyper IgE Syndrome, Autosomal Dominant
- HIES, Autosomal Dominant
- Autosomal Dominant HIES
- Autosomal Dominant HIESs
- HIESs, Autosomal Dominant
|
Below are MeSH descriptors whose meaning is more general than "Job Syndrome".
Below are MeSH descriptors whose meaning is more specific than "Job Syndrome".
This graph shows the total number of publications written about "Job Syndrome" by people in this website by year, and whether "Job Syndrome" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2001 | 1 | 0 | 1 |
| 2008 | 1 | 0 | 1 |
| 2014 | 0 | 1 | 1 |
| 2015 | 1 | 0 | 1 |
| 2016 | 1 | 0 | 1 |
| 2017 | 1 | 0 | 1 |
| 2019 | 1 | 0 | 1 |
| 2020 | 2 | 0 | 2 |
| 2021 | 1 | 0 | 1 |
| 2022 | 1 | 0 | 1 |
| 2024 | 1 | 0 | 1 |
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click here.
Below are the most recent publications written about "Job Syndrome" by people in Profiles.
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Allergic fungal rhinosinusitis linked to other hyper-IgE syndromes through defective TH17 responses. J Allergy Clin Immunol. 2024 Nov; 154(5):1169-1179.
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Recurrent Breast Abscesses in a Female with Autosomal Dominant Hyper-IgE Syndrome. J Clin Immunol. 2022 05; 42(4):889-891.
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Functional and structural analysis of cytokine-selective IL6ST defects that cause recessive hyper-IgE syndrome. J Allergy Clin Immunol. 2021 08; 148(2):585-598.
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Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome. J Exp Med. 2020 06 01; 217(6).
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Orthognathic Surgical Treatment in a Patient With Hyperimmunoglobulin E Syndrome. J Craniofac Surg. 2020 May/Jun; 31(3):e251-e254.
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Esophageal Intramural Pseudodiverticulosis With Tracking in a Child With Autosomal Dominant Hyper-IgE Syndrome. J Pediatr Gastroenterol Nutr. 2019 07; 69(1):e19.
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Gastrointestinal Manifestations of STAT3-Deficient Hyper-IgE Syndrome. J Clin Immunol. 2017 Oct; 37(7):695-700.
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Protein stabilization improves STAT3 function in autosomal dominant hyper-IgE syndrome. Blood. 2016 12 29; 128(26):3061-3072.
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The Ying and Yang of STAT3 in Human Disease. J Clin Immunol. 2015 Oct; 35(7):615-23.
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Transcription of the activating receptor NKG2D in natural killer cells is regulated by STAT3 tyrosine phosphorylation. Blood. 2014 Jul 17; 124(3):403-11.