"Lung Injury" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Damage to any compartment of the lung caused by physical, chemical, or biological agents which characteristically elicit inflammatory reaction. These inflammatory reactions can either be acute and dominated by NEUTROPHILS, or chronic and dominated by LYMPHOCYTES and MACROPHAGES.
| Descriptor ID |
D055370
|
| MeSH Number(s) |
C08.381.520 C26.891.554
|
| Concept/Terms |
Lung Injury- Lung Injury
- Injuries, Lung
- Injury, Lung
- Pulmonary Injury
- Injuries, Pulmonary
- Injury, Pulmonary
- Pulmonary Injuries
- Lung Injuries
Chronic Lung Injury- Chronic Lung Injury
- Chronic Lung Injuries
- Lung Injuries, Chronic
- Lung Injury, Chronic
|
Below are MeSH descriptors whose meaning is more general than "Lung Injury".
Below are MeSH descriptors whose meaning is more specific than "Lung Injury".
This graph shows the total number of publications written about "Lung Injury" by people in this website by year, and whether "Lung Injury" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 2 | 0 | 2 |
| 1996 | 0 | 1 | 1 |
| 1998 | 1 | 2 | 3 |
| 1999 | 0 | 1 | 1 |
| 2002 | 1 | 0 | 1 |
| 2003 | 2 | 1 | 3 |
| 2004 | 1 | 0 | 1 |
| 2005 | 1 | 1 | 2 |
| 2006 | 1 | 2 | 3 |
| 2008 | 4 | 1 | 5 |
| 2009 | 3 | 0 | 3 |
| 2010 | 1 | 1 | 2 |
| 2011 | 2 | 1 | 3 |
| 2012 | 1 | 1 | 2 |
| 2013 | 2 | 1 | 3 |
| 2014 | 2 | 1 | 3 |
| 2015 | 5 | 1 | 6 |
| 2016 | 3 | 2 | 5 |
| 2017 | 4 | 2 | 6 |
| 2018 | 4 | 3 | 7 |
| 2019 | 2 | 3 | 5 |
| 2020 | 6 | 1 | 7 |
| 2021 | 2 | 0 | 2 |
| 2022 | 4 | 0 | 4 |
| 2023 | 4 | 0 | 4 |
| 2024 | 2 | 1 | 3 |
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Below are the most recent publications written about "Lung Injury" by people in Profiles.
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TR? activation confers AT2-to-AT1 cell differentiation and anti-fibrosis during lung repair via KLF2 and CEBPA. Nat Commun. 2024 Oct 07; 15(1):8672.
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Mitochondrial (mt)DNA-cyclic GMP-AMP synthase (cGAS)-stimulator of interferon genes (STING) signaling promotes pyroptosis of macrophages via interferon regulatory factor (IRF)7/IRF3 activation to aggravate lung injury during severe acute pancreatitis. Cell Mol Biol Lett. 2024 Apr 27; 29(1):61.
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Necroptosis blockade prevents lung injury in severe influenza. Nature. 2024 Apr; 628(8009):835-843.
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Prenatal exposure to polycyclic aromatic hydrocarbons (PAHs) augments neonatal hyperoxic lung injury: Role of cytochrome P450 (CYP)1A1, 1A2, and 1B1. Free Radic Biol Med. 2024 02 01; 211:35-46.
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Loss of microRNA-30a and sex-specific effects on the neonatal hyperoxic lung injury. Biol Sex Differ. 2023 08 08; 14(1):50.
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Loss of growth differentiation factor 15 exacerbates lung injury in neonatal mice. Am J Physiol Lung Cell Mol Physiol. 2023 09 01; 325(3):L314-L326.
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Loss of cytochrome P450 (CYP)1B1 mitigates hyperoxia response in adult mouse lung by reprogramming metabolism and translation. Redox Biol. 2023 08; 64:102790.
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Allogeneic cord blood regulatory T cells can resolve lung inflammation. Cytotherapy. 2023 03; 25(3):245-253.
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Fulminant lung fibrosis in non-resolvable COVID-19 requiring transplantation. EBioMedicine. 2022 Dec; 86:104351.
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Remarkable sex-specific differences at single-cell resolution in neonatal hyperoxic lung injury. Am J Physiol Lung Cell Mol Physiol. 2023 01 01; 324(1):L5-L31.