Lymphocytic Choriomeningitis
"Lymphocytic Choriomeningitis" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A form of meningitis caused by LYMPHOCYTIC CHORIOMENINGITIS VIRUS. MICE and other rodents serve as the natural hosts, and infection in humans usually occurs through inhalation or ingestion of infectious particles. Clinical manifestations include an influenza-like syndrome followed by stiff neck, alterations of mentation, ATAXIA, and incontinence. Maternal infections may result in fetal malformations and injury, including neonatal HYDROCEPHALUS, aqueductal stenosis, CHORIORETINITIS, and MICROCEPHALY. (From Joynt, Clinical Neurology, 1996, Ch26, pp1-3)
| Descriptor ID |
D008216
|
| MeSH Number(s) |
C02.182.550.500 C02.782.082.580 C10.228.228.245.500.500 C10.228.614.400.500
|
| Concept/Terms |
Lymphocytic Choriomeningitis- Lymphocytic Choriomeningitis
- Choriomeningitis, Lymphocytic
- Lymphocytic Choriomeningitis Virus Encephalomyelitis
- Encephalomyelitis, Lymphocytic Choriomeningitis Virus
- Armstrong Syndrome
- Syndrome, Armstrong
- Armstrong's Syndrome
- Syndrome, Armstrong's
|
Below are MeSH descriptors whose meaning is more general than "Lymphocytic Choriomeningitis".
Below are MeSH descriptors whose meaning is more specific than "Lymphocytic Choriomeningitis".
This graph shows the total number of publications written about "Lymphocytic Choriomeningitis" by people in this website by year, and whether "Lymphocytic Choriomeningitis" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2004 | 0 | 1 | 1 |
| 2017 | 1 | 0 | 1 |
| 2018 | 1 | 0 | 1 |
| 2019 | 2 | 0 | 2 |
| 2020 | 1 | 1 | 2 |
| 2022 | 2 | 1 | 3 |
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Below are the most recent publications written about "Lymphocytic Choriomeningitis" by people in Profiles.
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Longitudinal Analysis of the Phenotype, Transcriptional Profile, and Anatomic Location of Memory CD8 T Cell Subsets after Acute Viral Infection. J Virol. 2023 01 31; 97(1):e0155622.
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PD-1 combination therapy with IL-2 modifies CD8+ T cell exhaustion program. Nature. 2022 10; 610(7930):173-181.
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TGF-? regulates the stem-like state of PD-1+ TCF-1+ virus-specific CD8 T cells during chronic infection. J Exp Med. 2022 10 03; 219(10).
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JAK/STAT pathway inhibition sensitizes CD8 T cells to dexamethasone-induced apoptosis in hyperinflammation. Blood. 2020 08 06; 136(6):657-668.
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PD-1+ stemlike CD8 T cells are resident in lymphoid tissues during persistent LCMV infection. Proc Natl Acad Sci U S A. 2020 02 25; 117(8):4292-4299.
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Proliferating Transitory T Cells with an Effector-like Transcriptional Signature Emerge from PD-1+ Stem-like CD8+ T Cells during Chronic Infection. Immunity. 2019 12 17; 51(6):1043-1058.e4.
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Expression of novel long noncoding RNAs defines virus-specific effector and memory CD8+ T cells. Nat Commun. 2019 01 14; 10(1):196.
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IL-33 induces immunosuppressive neutrophils via a type 2 innate lymphoid cell/IL-13/STAT6 axis and protects the liver against injury in LCMV infection-induced viral hepatitis. Cell Mol Immunol. 2019 02; 16(2):126-137.
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MicroRNA-22 controls interferon alpha production and erythroid maturation in response to infectious stress in mice. Exp Hematol. 2017 12; 56:7-15.
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IL-7-producing stromal cells are critical for lymph node remodeling. Blood. 2012 Dec 06; 120(24):4675-83.