Mitochondrial Proton-Translocating ATPases
"Mitochondrial Proton-Translocating ATPases" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Proton-translocating ATPases responsible for ADENOSINE TRIPHOSPHATE synthesis in the MITOCHONDRIA. They derive energy from the respiratory chain-driven reactions that develop high concentrations of protons within the intermembranous space of the mitochondria.
| Descriptor ID |
D025261
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| MeSH Number(s) |
D08.811.277.040.025.325.750 D08.811.913.696.650.150.500.750 D12.776.157.530.450.250.875.500.750 D12.776.543.585.450.250.875.500.750 D12.776.543.585.475.625 D12.776.575.750.625
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| Concept/Terms |
Mitochondrial Proton-Translocating ATPases- Mitochondrial Proton-Translocating ATPases
- ATPases, Mitochondrial Proton-Translocating
- Mitochondrial Proton Translocating ATPases
- Proton-Translocating ATPases, Mitochondrial
- Mitochondrial F(1)F(0) ATPase
- Mitochondrial ATP Synthase
- ATP Synthase, Mitochondrial
- Mitochondrial ATP Synthases
- ATP Synthases, Mitochondrial
|
Below are MeSH descriptors whose meaning is more general than "Mitochondrial Proton-Translocating ATPases".
Below are MeSH descriptors whose meaning is more specific than "Mitochondrial Proton-Translocating ATPases".
This graph shows the total number of publications written about "Mitochondrial Proton-Translocating ATPases" by people in this website by year, and whether "Mitochondrial Proton-Translocating ATPases" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2002 | 0 | 1 | 1 |
| 2004 | 1 | 1 | 2 |
| 2009 | 1 | 1 | 2 |
| 2010 | 0 | 1 | 1 |
| 2013 | 1 | 0 | 1 |
| 2014 | 1 | 0 | 1 |
| 2017 | 1 | 0 | 1 |
| 2018 | 4 | 0 | 4 |
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Below are the most recent publications written about "Mitochondrial Proton-Translocating ATPases" by people in Profiles.
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The mitochondrial DNA variant m.9032T?>?C in MT-ATP6 encoding p.(Leu169Pro) causes a complex mitochondrial neurological syndrome. Mitochondrion. 2020 11; 55:8-13.
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Mitochondrial Genome Variation Affects Multiple Respiration and Nonrespiration Phenotypes in Saccharomyces cerevisiae. Genetics. 2019 02; 211(2):773-786.
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Mitochondrial reprogramming via ATP5H loss promotes multimodal cancer therapy resistance. J Clin Invest. 2018 08 31; 128(9):4098-4114.
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Novel insights into the functional metabolic impact of an apparent de novo m.8993T>G variant in the MT-ATP6 gene associated with maternally inherited form of Leigh Syndrome. Mol Genet Metab. 2018 05; 124(1):71-81.
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Biallelic Mutations in ATP5F1D, which Encodes a Subunit of ATP Synthase, Cause a Metabolic Disorder. Am J Hum Genet. 2018 03 01; 102(3):494-504.
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Clinical, biochemical, and genetic features associated with VARS2-related mitochondrial disease. Hum Mutat. 2018 04; 39(4):563-578.
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Biochemical signatures mimicking multiple carboxylase deficiency in children with mutations in MT-ATP6. Mitochondrion. 2019 01; 44:58-64.
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Involvement of FANCD2 in Energy Metabolism via ATP5a. Sci Rep. 2017 07 07; 7(1):4921.
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Mitochondrial myopathy, lactic acidosis, and sideroblastic anemia (MLASA) plus associated with a novel de novo mutation (m.8969G>A) in the mitochondrial encoded ATP6 gene. Mol Genet Metab. 2014 Nov; 113(3):207-12.
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How the nucleus and mitochondria communicate in energy production during stress: nuclear MtATP6, an early-stress responsive gene, regulates the mitochondrial F1F0-ATP synthase complex. Mol Biotechnol. 2013 Jul; 54(3):756-69.