"Mitochondria" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Semiautonomous, self-reproducing organelles that occur in the cytoplasm of all cells of most, but not all, eukaryotes. Each mitochondrion is surrounded by a double limiting membrane. The inner membrane is highly invaginated, and its projections are called cristae. Mitochondria are the sites of the reactions of oxidative phosphorylation, which result in the formation of ATP. They contain distinctive RIBOSOMES, transfer RNAs (RNA, TRANSFER); AMINO ACYL T RNA SYNTHETASES; and elongation and termination factors. Mitochondria depend upon genes within the nucleus of the cells in which they reside for many essential messenger RNAs (RNA, MESSENGER). Mitochondria are believed to have arisen from aerobic bacteria that established a symbiotic relationship with primitive protoeukaryotes. (King & Stansfield, A Dictionary of Genetics, 4th ed)
| Descriptor ID |
D008928
|
| MeSH Number(s) |
A11.284.430.214.190.875.564 A11.284.835.626
|
| Concept/Terms |
Mitochondrial Contraction- Mitochondrial Contraction
- Contraction, Mitochondrial
- Contractions, Mitochondrial
- Mitochondrial Contractions
|
Below are MeSH descriptors whose meaning is more general than "Mitochondria".
Below are MeSH descriptors whose meaning is more specific than "Mitochondria".
This graph shows the total number of publications written about "Mitochondria" by people in this website by year, and whether "Mitochondria" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1996 | 3 | 0 | 3 |
| 1997 | 2 | 0 | 2 |
| 1998 | 1 | 2 | 3 |
| 1999 | 4 | 5 | 9 |
| 2000 | 3 | 2 | 5 |
| 2001 | 2 | 1 | 3 |
| 2002 | 3 | 3 | 6 |
| 2003 | 5 | 5 | 10 |
| 2004 | 8 | 4 | 12 |
| 2005 | 7 | 3 | 10 |
| 2006 | 4 | 7 | 11 |
| 2007 | 1 | 11 | 12 |
| 2008 | 3 | 3 | 6 |
| 2009 | 9 | 5 | 14 |
| 2010 | 11 | 6 | 17 |
| 2011 | 11 | 7 | 18 |
| 2012 | 11 | 9 | 20 |
| 2013 | 8 | 11 | 19 |
| 2014 | 14 | 6 | 20 |
| 2015 | 20 | 7 | 27 |
| 2016 | 19 | 13 | 32 |
| 2017 | 13 | 12 | 25 |
| 2018 | 14 | 16 | 30 |
| 2019 | 11 | 11 | 22 |
| 2020 | 21 | 16 | 37 |
| 2021 | 15 | 20 | 35 |
| 2022 | 2 | 13 | 15 |
| 2023 | 9 | 13 | 22 |
| 2024 | 7 | 15 | 22 |
| 2025 | 22 | 14 | 36 |
| 2026 | 9 | 3 | 12 |
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Below are the most recent publications written about "Mitochondria" by people in Profiles.
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Measuring Calcium Efflux in Isolated Primary Mitochondria. Curr Protoc. 2026 Jun; 6(6):e70393.
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Brain Bioenergetics in Aging: Neurovascular and Neurometabolic Coupling and Fuels: 15th International Conference on Brain Energy Metabolism. J Neurochem. 2026 Jun; 170(6):e70467.
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The MICOS Complex Regulates Mitochondrial Structure and Oxidative Stress During Age-Dependent Structural Deficits in the Kidney. Aging Cell. 2026 May; 25(5):e70534.
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Clotting the Gap Between Mitochondria-Mediated Immunity and Mitochondrial Transfer. Circ Res. 2026 Apr 10; 138(8):e326987.
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Regulation of mitochondrial ROS by C15ORF48 in a basal cell subpopulation contributes to chemotherapy resistance in TNBC. Sci Adv. 2026 Apr 03; 12(14):eaec8684.
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Th17 cells require the DNA repair sensor xeroderma pigmentosum complementation Group C to control oxidative DNA damage in a murine model. Nat Commun. 2026 Apr 01; 17(1).
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High-fat diet causes rapid loss of intestinal group 3 innate lymphoid cells through microbiota-driven inflammation and mitochondrial stress. Immunity. 2026 Apr 14; 59(4):988-1005.e9.
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CRISPR-Cas-based activation of PPARGC1A boosts endogenous mitochondria and enhances cardiac function after myocardial infarction. Mol Ther. 2026 Jun 03; 34(6):3320-3333.
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a-synuclein preformed fibrils suppress cell cycle progression and glycolytic flux in glioblastoma cells. Exp Cell Res. 2026 Apr 15; 457(2):114970.
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Synergistic Effects of Honokiol on Camptothecin-triggered Apoptosis of Drug-resistant Glioblastoma Cells via CDK1-mediated G2/M Arrest and Intrinsic ROS-Mitochondrion-dependent Pathway. Anticancer Res. 2026 Feb; 46(2):633-649.