Cyclin-Dependent Kinase Inhibitor p19
"Cyclin-Dependent Kinase Inhibitor p19" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An INK4 cyclin-dependent kinase inhibitor containing five ANKYRIN REPEATS. Aberrant expression of this protein has been associated with TESTICULAR CANCER.
Descriptor ID |
D050764
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MeSH Number(s) |
D12.644.360.225.400 D12.776.167.187.400 D12.776.476.225.400 D12.776.624.776.355.400
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Concept/Terms |
Cyclin-Dependent Kinase Inhibitor p19- Cyclin-Dependent Kinase Inhibitor p19
- Cyclin Dependent Kinase Inhibitor p19
- INK4D Protein
- p19INK4D Protein
- Cyclin-Dependent Kinase Inhibitor 2D
- Cyclin Dependent Kinase Inhibitor 2D
- CDKN2D Protein
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Below are MeSH descriptors whose meaning is more general than "Cyclin-Dependent Kinase Inhibitor p19".
Below are MeSH descriptors whose meaning is more specific than "Cyclin-Dependent Kinase Inhibitor p19".
This graph shows the total number of publications written about "Cyclin-Dependent Kinase Inhibitor p19" by people in this website by year, and whether "Cyclin-Dependent Kinase Inhibitor p19" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2009 | 0 | 1 | 1 |
2012 | 0 | 1 | 1 |
2014 | 1 | 0 | 1 |
2015 | 0 | 1 | 1 |
2017 | 0 | 1 | 1 |
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Below are the most recent publications written about "Cyclin-Dependent Kinase Inhibitor p19" by people in Profiles.
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Role of CDKN2C Fluorescence In Situ Hybridization in the Management of Medullary Thyroid Carcinoma. Ann Clin Lab Sci. 2017 Sep; 47(5):523-528.
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Limited mitochondrial permeabilization causes DNA damage and genomic instability in the absence of cell death. Mol Cell. 2015 Mar 05; 57(5):860-872.
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CDKN2D-WDFY2 is a cancer-specific fusion gene recurrent in high-grade serous ovarian carcinoma. PLoS Genet. 2014 Mar; 10(3):e1004216.
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SINE retrotransposons cause epigenetic reprogramming of adjacent gene promoters. Mol Cancer Res. 2012 Oct; 10(10):1332-42.
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Inhibition of NOTCH signaling by gamma secretase inhibitor engages the RB pathway and elicits cell cycle exit in T-cell acute lymphoblastic leukemia cells. Cancer Res. 2009 Apr 01; 69(7):3060-8.