Mitogen-Activated Protein Kinase 9
"Mitogen-Activated Protein Kinase 9" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A c-jun amino-terminal kinase that is activated by environmental stress and pro-inflammatory cytokines. Several isoforms of the protein with molecular sizes of 48 and 54 KD exist due to multiple ALTERNATIVE SPLICING.
| Descriptor ID |
D048056
|
| MeSH Number(s) |
D08.811.913.696.620.682.700.567.374.750 D12.644.360.450.340.750 D12.776.476.450.340.750
|
| Concept/Terms |
Mitogen-Activated Protein Kinase 9- Mitogen-Activated Protein Kinase 9
- Mitogen Activated Protein Kinase 9
- Stress-Activated Protein Kinase JNK2
- Stress Activated Protein Kinase JNK2
- JNK2 Kinase
- c-jun Kinase-2
- Kinase-2, c-jun
- c jun Kinase 2
- MAPK9 Mitogen-Activated Protein Kinase
- MAPK9 Mitogen Activated Protein Kinase
- c-jun N-terminal Kinase 2
- c jun N terminal Kinase 2
- JNK2 Stress-Activated Protein Kinase
- JNK2 Stress Activated Protein Kinase
|
Below are MeSH descriptors whose meaning is more general than "Mitogen-Activated Protein Kinase 9".
Below are MeSH descriptors whose meaning is more specific than "Mitogen-Activated Protein Kinase 9".
This graph shows the total number of publications written about "Mitogen-Activated Protein Kinase 9" by people in this website by year, and whether "Mitogen-Activated Protein Kinase 9" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2006 | 1 | 0 | 1 |
| 2008 | 0 | 1 | 1 |
| 2009 | 3 | 0 | 3 |
| 2011 | 1 | 0 | 1 |
| 2013 | 0 | 1 | 1 |
| 2019 | 1 | 0 | 1 |
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Below are the most recent publications written about "Mitogen-Activated Protein Kinase 9" by people in Profiles.
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JNK1/2 represses Lkb1-deficiency-induced lung squamous cell carcinoma progression. Nat Commun. 2019 05 14; 10(1):2148.
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Differential regulation of c-Jun protein plays an instrumental role in chemoresistance of cancer cells. J Biol Chem. 2013 Jul 05; 288(27):19321-9.
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Low humidity environmental challenge causes barrier disruption and cornification of the mouse corneal epithelium via a c-jun N-terminal kinase 2 (JNK2) pathway. Exp Eye Res. 2012 Jan; 94(1):150-6.
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c-Jun N-terminal kinase 2 (JNK2) enhances cell migration through epidermal growth factor substrate 8 (EPS8). J Biol Chem. 2011 Apr 29; 286(17):15287-97.
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Essential role for c-Jun N-terminal kinase 2 in corneal epithelial response to desiccating stress. Arch Ophthalmol. 2009 Dec; 127(12):1625-31.
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Redundant roles for cJun-N-terminal kinase 1 and 2 in interleukin-1beta-mediated reduction and modification of murine hepatic nuclear retinoid X receptor alpha. J Hepatol. 2009 Nov; 51(5):898-908.
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The JNKs differentially regulate RNA polymerase III transcription by coordinately modulating the expression of all TFIIIB subunits. Proc Natl Acad Sci U S A. 2009 Aug 04; 106(31):12682-7.
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JNK-deficiency enhanced oncolytic vaccinia virus replication and blocked activation of double-stranded RNA-dependent protein kinase. Cancer Gene Ther. 2008 Sep; 15(9):616-24.
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Hyperosmolarity-induced cornification of human corneal epithelial cells is regulated by JNK MAPK. Invest Ophthalmol Vis Sci. 2008 Feb; 49(2):539-49.
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Proposals and rationale for revision of the World Health Organization diagnostic criteria for polycythemia vera, essential thrombocythemia, and primary myelofibrosis: recommendations from an ad hoc international expert panel. Blood. 2007 Aug 15; 110(4):1092-7.