Antigens, Polyomavirus Transforming
"Antigens, Polyomavirus Transforming" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Polyomavirus antigens which cause infection and cellular transformation. The large T antigen is necessary for the initiation of viral DNA synthesis, repression of transcription of the early region and is responsible in conjunction with the middle T antigen for the transformation of primary cells. Small T antigen is necessary for the completion of the productive infection cycle.
Descriptor ID |
D000952
|
MeSH Number(s) |
D12.776.624.664.520.090 D12.776.964.700.090 D23.050.285.062.090 D23.050.327.062.090
|
Concept/Terms |
Antigens, Polyomavirus Transforming- Antigens, Polyomavirus Transforming
- Polyomavirus Tumor Antigens
- Antigens, Polyomavirus Tumor
- Tumor Antigens, Polyomavirus
- Polyomavirus Transforming Antigens
- Transforming Antigens, Polyomavirus
SV40 T Proteins- SV40 T Proteins
- Proteins, SV40 T
- T Proteins, SV40
- SV40 T Antigens
- Antigens, SV40 T
- T Antigens, SV40
|
Below are MeSH descriptors whose meaning is more general than "Antigens, Polyomavirus Transforming".
- Chemicals and Drugs [D]
- Amino Acids, Peptides, and Proteins [D12]
- Proteins [D12.776]
- Neoplasm Proteins [D12.776.624]
- Oncogene Proteins [D12.776.624.664]
- Oncogene Proteins, Viral [D12.776.624.664.520]
- Antigens, Polyomavirus Transforming [D12.776.624.664.520.090]
- Viral Proteins [D12.776.964]
- Oncogene Proteins, Viral [D12.776.964.700]
- Antigens, Polyomavirus Transforming [D12.776.964.700.090]
- Biological Factors [D23]
- Antigens [D23.050]
- Antigens, Neoplasm [D23.050.285]
- Antigens, Viral, Tumor [D23.050.285.062]
- Antigens, Polyomavirus Transforming [D23.050.285.062.090]
- Antigens, Viral [D23.050.327]
- Antigens, Viral, Tumor [D23.050.327.062]
- Antigens, Polyomavirus Transforming [D23.050.327.062.090]
Below are MeSH descriptors whose meaning is more specific than "Antigens, Polyomavirus Transforming".
This graph shows the total number of publications written about "Antigens, Polyomavirus Transforming" by people in this website by year, and whether "Antigens, Polyomavirus Transforming" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
Year | Major Topic | Minor Topic | Total |
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1994 | 2 | 1 | 3 |
1995 | 0 | 1 | 1 |
1996 | 2 | 1 | 3 |
1997 | 5 | 0 | 5 |
1998 | 3 | 2 | 5 |
1999 | 0 | 3 | 3 |
2000 | 3 | 5 | 8 |
2001 | 3 | 0 | 3 |
2002 | 3 | 1 | 4 |
2003 | 0 | 4 | 4 |
2004 | 3 | 3 | 6 |
2005 | 2 | 2 | 4 |
2006 | 1 | 3 | 4 |
2007 | 1 | 3 | 4 |
2008 | 1 | 2 | 3 |
2009 | 2 | 4 | 6 |
2011 | 0 | 1 | 1 |
2012 | 1 | 1 | 2 |
2013 | 2 | 1 | 3 |
2014 | 1 | 2 | 3 |
2018 | 0 | 1 | 1 |
To return to the timeline,
click here.
Below are the most recent publications written about "Antigens, Polyomavirus Transforming" by people in Profiles.
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Viral microRNA effects on persistent infection of human lymphoid cells by polyomavirus SV40. PLoS One. 2018; 13(2):e0192799.
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The piggyBac transposon-mediated expression of SV40 T antigen efficiently immortalizes mouse embryonic fibroblasts (MEFs). PLoS One. 2014; 9(5):e97316.
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Regulation of Rad51 promoter. Cell Cycle. 2014; 13(13):2038-45.
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Regression of metastatic Merkel cell carcinoma following transfer of polyomavirus-specific T cells and therapies capable of re-inducing HLA class-I. Cancer Immunol Res. 2014 Jan; 2(1):27-36.
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Efficient delivery of miR-122 to regulate cholesterol metabolism using a non-covalent peptide-based strategy. Mol Med Rep. 2013 Nov; 8(5):1472-8.
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Can SV40 infect and immortalize human B-lymphocytes and mesothelial cells as a natural pathogen? Leuk Res. 2013 Jun; 37(6):607-8.
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Aire-dependent thymic development of tumor-associated regulatory T cells. Science. 2013 Mar 08; 339(6124):1219-24.
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Embryonic retinal tumors in SV40 T-Ag transgenic mice contain CD133+ tumor-initiating cells. Invest Ophthalmol Vis Sci. 2012 Jun 08; 53(7):3454-62.
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Mucinous adenocarcinoma developed from human fallopian tube epithelial cells through defined genetic modifications. Cell Cycle. 2012 Jun 01; 11(11):2107-13.
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Characterization of the oncogenic activity of the novel TRIM59 gene in mouse cancer models. Mol Cancer Ther. 2011 Jul; 10(7):1229-40.