"Multivesicular Bodies" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Endosomes containing intraluminal vesicles which are formed by the inward budding of the endosome membrane. Multivesicular bodies (MVBs) may fuse with other organelles such as LYSOSOMES or fuse back with the PLASMA MEMBRANE releasing their contents by EXOCYTOSIS. The MVB intraluminal vesicles released into the extracellular environment are known as EXOSOMES.
| Descriptor ID |
D057146
|
| MeSH Number(s) |
A11.284.430.214.190.875.190.880.337.500
|
| Concept/Terms |
Multivesicular Bodies- Multivesicular Bodies
- Multivesicular Body
- Endosomal Carrier Vesicles
- Endosomal Carrier Vesicle
- Multi-Vesicular Bodies
- Multi Vesicular Bodies
- Multi-Vesicular Body
|
Below are MeSH descriptors whose meaning is more general than "Multivesicular Bodies".
Below are MeSH descriptors whose meaning is more specific than "Multivesicular Bodies".
This graph shows the total number of publications written about "Multivesicular Bodies" by people in this website by year, and whether "Multivesicular Bodies" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2018 | 0 | 2 | 2 |
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Below are the most recent publications written about "Multivesicular Bodies" by people in Profiles.
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RAB31 marks and controls an ESCRT-independent exosome pathway. Cell Res. 2021 02; 31(2):157-177.
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A non-canonical autophagy-dependent role of the ATG16L1T300A variant in urothelial vesicular trafficking and uropathogenic Escherichia coli persistence. Autophagy. 2019 03; 15(3):527-542.
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Distinct mechanisms enable inward or outward budding from late endosomes/multivesicular bodies. Exp Cell Res. 2018 11 01; 372(1):1-15.
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Inhibition of endoplasmic-reticulum-stress-mediated autophagy enhances the effectiveness of chemotherapeutics on pancreatic cancer. J Transl Med. 2018 07 09; 16(1):190.
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Phosphorylation-Dependent Activation of the ESCRT Function of ALIX in Cytokinetic Abscission and Retroviral Budding. Dev Cell. 2016 Feb 08; 36(3):331-43.
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Unravelling the pivotal role of Alix in MVB sorting and silencing of the activated EGFR. Biochem J. 2015 Mar 15; 466(3):475-87.
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Crimean-Congo hemorrhagic fever virus entry into host cells occurs through the multivesicular body and requires ESCRT regulators. PLoS Pathog. 2014 Sep; 10(9):e1004390.