"Marine Toxins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Toxic or poisonous substances elaborated by marine flora or fauna. They include also specific, characterized poisons or toxins for which there is no more specific heading, like those from poisonous FISHES.
| Descriptor ID |
D008387
|
| MeSH Number(s) |
D23.946.580
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Marine Toxins".
Below are MeSH descriptors whose meaning is more specific than "Marine Toxins".
This graph shows the total number of publications written about "Marine Toxins" by people in this website by year, and whether "Marine Toxins" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1997 | 0 | 2 | 2 |
| 2007 | 0 | 1 | 1 |
| 2014 | 0 | 1 | 1 |
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Below are the most recent publications written about "Marine Toxins" by people in Profiles.
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Quantitation of myosin regulatory light chain phosphorylation in biological samples with multiple reaction monitoring mass spectrometry. Biochim Biophys Acta Proteins Proteom. 2018 May - Jun; 1866(5-6):608-616.
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Channel-anchored protein kinase CK2 and protein phosphatase 1 reciprocally regulate KCNQ2-containing M-channels via phosphorylation of calmodulin. J Biol Chem. 2014 Apr 18; 289(16):11536-11544.
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Cyclic AMP-independent CGRP8-37-sensitive receptors mediate adrenomedullin-induced decrease of CaCl2-contraction in pregnant rat mesenteric artery. J Vasc Res. 2008; 45(1):33-44.
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Mediation of Sonic hedgehog-induced expression of COUP-TFII by a protein phosphatase. Science. 1997 Dec 12; 278(5345):1947-50.
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Troponin I phosphorylation in spontaneously hypertensive rat heart: effect of beta-adrenergic stimulation. Am J Physiol. 1997 Sep; 273(3 Pt 2):H1440-51.
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The serine/threonine phosphatase inhibitor calyculin A induces rapid degradation of IkappaBbeta. Requirement of both the N- and C-terminal sequences. J Biol Chem. 1997 Feb 28; 272(9):5409-12.
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Multiple structural domains within I kappa B alpha are required for its inducible degradation by both cytokines and phosphatase inhibitors. Biochem Biophys Res Commun. 1996 Jun 05; 223(1):123-8.
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Reversible protein phosphorylation modulates nucleotide excision repair of damaged DNA by human cell extracts. Nucleic Acids Res. 1996 Feb 01; 24(3):433-40.
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Activation of NF-kappa B by phosphatase inhibitors involves the phosphorylation of I kappa B alpha at phosphatase 2A-sensitive sites. J Biol Chem. 1995 Aug 04; 270(31):18347-51.