"Endometrial Hyperplasia" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Benign proliferation of the ENDOMETRIUM in the UTERUS. Endometrial hyperplasia is classified by its cytology and glandular tissue. There are simple, complex (adenomatous without atypia), and atypical hyperplasia representing also the ascending risk of becoming malignant.
| Descriptor ID |
D004714
|
| MeSH Number(s) |
C13.351.500.852.228
|
| Concept/Terms |
Endometrial Hyperplasia- Endometrial Hyperplasia
- Endometrial Hyperplasias
- Hyperplasia, Endometrial
- Hyperplasias, Endometrial
Atypical Endometrial Hyperplasia- Atypical Endometrial Hyperplasia
- Atypical Endometrial Hyperplasias
- Endometrial Hyperplasia, Atypical
- Endometrial Hyperplasias, Atypical
- Hyperplasia, Atypical Endometrial
- Hyperplasias, Atypical Endometrial
Complex Endometrial Hyperplasia- Complex Endometrial Hyperplasia
- Complex Endometrial Hyperplasias
- Endometrial Hyperplasia, Complex
- Endometrial Hyperplasias, Complex
- Hyperplasia, Complex Endometrial
- Hyperplasias, Complex Endometrial
Simple Endometrial Hyperplasia- Simple Endometrial Hyperplasia
- Endometrial Hyperplasia, Simple
- Endometrial Hyperplasias, Simple
- Hyperplasia, Simple Endometrial
- Hyperplasias, Simple Endometrial
- Simple Endometrial Hyperplasias
|
Below are MeSH descriptors whose meaning is more general than "Endometrial Hyperplasia".
Below are MeSH descriptors whose meaning is more specific than "Endometrial Hyperplasia".
This graph shows the total number of publications written about "Endometrial Hyperplasia" by people in this website by year, and whether "Endometrial Hyperplasia" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2005 | 1 | 0 | 1 |
| 2008 | 2 | 0 | 2 |
| 2009 | 1 | 0 | 1 |
| 2010 | 2 | 0 | 2 |
| 2013 | 0 | 1 | 1 |
| 2014 | 1 | 2 | 3 |
| 2017 | 1 | 0 | 1 |
| 2018 | 1 | 0 | 1 |
| 2019 | 1 | 0 | 1 |
| 2025 | 1 | 0 | 1 |
| 2026 | 1 | 0 | 1 |
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click here.
Below are the most recent publications written about "Endometrial Hyperplasia" by people in Profiles.
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The role of metabolic and bariatric surgery for obesity management in endometrial intraepithelial neoplasia and endometrial cancer: A Society of Gynecologic Oncology and Society of American Gastrointestinal and Endoscopic Surgeons clinical practice statement. Gynecol Oncol. 2026 Sep; 212:31-42.
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TGFBR2 coordinates the endometrial response to estrogen, regulating endometrial hyperplasia and fertility. Proc Natl Acad Sci U S A. 2025 Dec 09; 122(49):e2518507122.
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Enhancer of Zeste 2 Polycomb Repressive Complex 2 Subunit Is Required for Uterine Epithelial Integrity. Am J Pathol. 2019 06; 189(6):1212-1225.
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Sonic hedgehog protects endometrial hyperplasial cells against oxidative stress via suppressing mitochondrial fission protein dynamin-like GTPase (Drp1). Free Radic Biol Med. 2018 12; 129:582-599.
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The regulation of Hh/Gli1 signaling cascade involves Gsk3?- mediated mechanism in estrogen-derived endometrial hyperplasia. Sci Rep. 2017 07 26; 7(1):6557.
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Clinically significant endometrial cancer risk following a diagnosis of complex atypical hyperplasia. Gynecol Oncol. 2014 Dec; 135(3):451-4.
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Inhibitory effect of 2-(piperidinoethoxyphenyl)-3-(4-hydroxyphenyl)-2H-benzo(b)pyran (K-1) on human primary endometrial hyperplasial cells mediated via combined suppression of Wnt/?-catenin signaling and PI3K/Akt survival pathway. Cell Death Dis. 2014 Aug 21; 5:e1380.
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A mouse model for endometrioid ovarian cancer arising from the distal oviduct. Int J Cancer. 2014 Sep 01; 135(5):1028-37.
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Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer. Cancer Res. 2013 Aug 15; 73(16):5090-9.
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Mig-6 modulates uterine steroid hormone responsiveness and exhibits altered expression in endometrial disease. Proc Natl Acad Sci U S A. 2009 May 26; 106(21):8677-82.