Epithelial Sodium Channels
"Epithelial Sodium Channels" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Sodium channels found on salt-reabsorbing EPITHELIAL CELLS that line the distal NEPHRON; the distal COLON; SALIVARY DUCTS; SWEAT GLANDS; and the LUNG. They are AMILORIDE-sensitive and play a critical role in the control of sodium balance, BLOOD VOLUME, and BLOOD PRESSURE.
| Descriptor ID |
D053503
|
| MeSH Number(s) |
D12.776.157.530.400.875.200 D12.776.543.550.450.875.200 D12.776.543.585.400.875.200
|
| Concept/Terms |
Epithelial Sodium Channels- Epithelial Sodium Channels
- Sodium Channels, Epithelial
- Epithelial Amiloride-Sensitive Sodium Channel
- Epithelial Amiloride Sensitive Sodium Channel
- Epithelial Sodium Ion Channels
- ENaC (Epithelial Na+ Channel)
- Epithelial Sodium Channel
- Sodium Channel, Epithelial
|
Below are MeSH descriptors whose meaning is more general than "Epithelial Sodium Channels".
Below are MeSH descriptors whose meaning is more specific than "Epithelial Sodium Channels".
This graph shows the total number of publications written about "Epithelial Sodium Channels" by people in this website by year, and whether "Epithelial Sodium Channels" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2005 | 0 | 1 | 1 |
| 2006 | 0 | 1 | 1 |
| 2007 | 0 | 1 | 1 |
| 2008 | 0 | 2 | 2 |
| 2009 | 1 | 0 | 1 |
| 2010 | 0 | 2 | 2 |
| 2012 | 0 | 1 | 1 |
| 2014 | 1 | 0 | 1 |
| 2016 | 1 | 0 | 1 |
| 2022 | 1 | 0 | 1 |
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Below are the most recent publications written about "Epithelial Sodium Channels" by people in Profiles.
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Rare Variants in Genes Encoding Subunits of the Epithelial Na+ Channel Are Associated With Blood Pressure and Kidney Function. Hypertension. 2022 11; 79(11):2573-2582.
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Whole-exome sequencing reveals an inherited R566X mutation of the epithelial sodium channel ?-subunit in a case of early-onset phenotype of Liddle syndrome. Cold Spring Harb Mol Case Stud. 2016 11; 2(6):a001255.
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Disease-targeted sequencing of ion channel genes identifies de novo mutations in patients with non-familial Brugada syndrome. Sci Rep. 2014 Oct 23; 4:6733.
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Genomic association analysis identifies multiple loci influencing antihypertensive response to an angiotensin II receptor blocker. Hypertension. 2012 Jun; 59(6):1204-11.
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Pharmacogenetic association of hypertension candidate genes with fasting glucose in the GenHAT Study. J Hypertens. 2010 Oct; 28(10):2076-83.
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Defective CFTR induces aggresome formation and lung inflammation in cystic fibrosis through ROS-mediated autophagy inhibition. Nat Cell Biol. 2010 Sep; 12(9):863-75.
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Identification of protein domains that control proton and calcium sensitivity of ASIC1a. J Biol Chem. 2009 Oct 09; 284(41):27899-27907.
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Lack of the serum and glucocorticoid-inducible kinase SGK1 attenuates the volume retention after treatment with the PPARgamma agonist pioglitazone. Pflugers Arch. 2008 May; 456(2):425-36.
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Amiloride-sensitive and amiloride-insensitive kaliuresis in advanced chronic kidney disease. J Nephrol. 2008 Jan-Feb; 21(1):93-8.
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17p11.2p12 triplication and del(17)q11.2q12 in a severely affected child with dup(17)p11.2p12 syndrome. Clin Genet. 2007 Jul; 72(1):47-58.