Calcification, Physiologic
"Calcification, Physiologic" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Process by which organic tissue becomes hardened by the physiologic deposit of calcium salts.
Descriptor ID |
D002113
|
MeSH Number(s) |
G07.345.186 G07.345.500.325.377.625.100.175 G11.427.578.100.175
|
Concept/Terms |
Calcification, Physiologic- Calcification, Physiologic
- Calcification, Physiological
- Physiological Calcification
- Physiologic Calcification
|
Below are MeSH descriptors whose meaning is more general than "Calcification, Physiologic".
Below are MeSH descriptors whose meaning is more specific than "Calcification, Physiologic".
This graph shows the total number of publications written about "Calcification, Physiologic" by people in this website by year, and whether "Calcification, Physiologic" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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1995 | 1 | 0 | 1 |
2000 | 1 | 0 | 1 |
2002 | 1 | 1 | 2 |
2004 | 1 | 0 | 1 |
2005 | 2 | 0 | 2 |
2006 | 0 | 1 | 1 |
2010 | 0 | 1 | 1 |
2012 | 0 | 1 | 1 |
2013 | 0 | 2 | 2 |
2014 | 2 | 2 | 4 |
2017 | 0 | 1 | 1 |
2019 | 0 | 1 | 1 |
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Below are the most recent publications written about "Calcification, Physiologic" by people in Profiles.
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Genetic and Pharmacological Targeting of Transcriptional Repression in Resistance to Thyroid Hormone Alpha. Thyroid. 2019 05; 29(5):726-734.
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Proteomics Profiling of Exosomes from Primary Mouse Osteoblasts under Proliferation versus Mineralization Conditions and Characterization of Their Uptake into Prostate Cancer Cells. J Proteome Res. 2017 08 04; 16(8):2709-2728.
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Fkbp10 Deletion in Osteoblasts Leads to Qualitative Defects in Bone. J Bone Miner Res. 2017 Jun; 32(6):1354-1367.
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Genetic causes and mechanisms of Osteogenesis Imperfecta. Bone. 2017 Sep; 102:40-49.
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Should monitoring of fibroblast growth factor-23 levels in dialysis patients be a part of routine clinical practice? Semin Dial. 2014 Nov-Dec; 27(6):565-8.
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Asfotase-a improves bone growth, mineralization and strength in mouse models of neurofibromatosis type-1. Nat Med. 2014 Aug; 20(8):904-10.
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Neurofibromin inactivation impairs osteocyte development in Nf1Prx1 and Nf1Col1 mouse models. Bone. 2014 Sep; 66:155-62.
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The loss of activating transcription factor 4 (ATF4) reduces bone toughness and fracture toughness. Bone. 2014 May; 62:1-9.
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Multiscale, converging defects of macro-porosity, microstructure and matrix mineralization impact long bone fragility in NF1. PLoS One. 2014; 9(1):e86115.
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Screen for abnormal mitochondrial phenotypes in mouse embryonic stem cells identifies a model for succinyl-CoA ligase deficiency and mtDNA depletion. Dis Model Mech. 2014 Feb; 7(2):271-80.