"Qa-SNARE Proteins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A subfamily of Q-SNARE PROTEINS which occupy the same position as syntaxin 1A in the SNARE complex and which also are most similar to syntaxin 1A in their AMINO ACID SEQUENCE. This subfamily is also known as the syntaxins, although a few so called syntaxins are Qc-SNARES.
| Descriptor ID |
D050765
|
| MeSH Number(s) |
D12.776.543.512.249.500.500 D12.776.543.990.775.500.500
|
| Concept/Terms |
Qa-SNARE Proteins- Qa-SNARE Proteins
- Qa SNARE Proteins
- Qa-SNAREs
- Qa SNAREs
- Syntaxin Proteins
- Proteins, Syntaxin
- Syntaxins
- Syntaxin Protein
- Protein, Syntaxin
- Syntaxin
- Syntaxin 1A Homologs
Syntaxin 11- Syntaxin 11
- Syntaxin 11 Protein
- Protein, Syntaxin 11
|
Below are MeSH descriptors whose meaning is more general than "Qa-SNARE Proteins".
Below are MeSH descriptors whose meaning is more specific than "Qa-SNARE Proteins".
This graph shows the total number of publications written about "Qa-SNARE Proteins" by people in this website by year, and whether "Qa-SNARE Proteins" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1995 | 0 | 1 | 1 |
| 1998 | 0 | 1 | 1 |
| 2001 | 0 | 2 | 2 |
| 2004 | 0 | 2 | 2 |
| 2005 | 1 | 0 | 1 |
| 2010 | 0 | 1 | 1 |
| 2014 | 1 | 0 | 1 |
| 2018 | 1 | 0 | 1 |
| 2019 | 0 | 1 | 1 |
| 2022 | 0 | 1 | 1 |
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Below are the most recent publications written about "Qa-SNARE Proteins" by people in Profiles.
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Long read sequencing and expression studies of AHDC1 deletions in Xia-Gibbs syndrome reveal a novel genetic regulatory mechanism. Hum Mutat. 2022 12; 43(12):2033-2053.
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Pathogenic STX3 variants affecting the retinal and intestinal transcripts cause an early-onset severe retinal dystrophy in microvillus inclusion disease subjects. Hum Genet. 2021 Aug; 140(8):1143-1156.
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Defining the layers of a sensory cilium with STORM and cryoelectron nanoscopy. Proc Natl Acad Sci U S A. 2019 11 19; 116(47):23562-23572.
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Syntaxin 3, but not syntaxin 4, is required for mast cell-regulated exocytosis, where it plays a primary role mediating compound exocytosis. J Biol Chem. 2019 03 01; 294(9):3012-3023.
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The syntaxin 31-induced gene, LESION SIMULATING DISEASE1 (LSD1), functions in Glycine max defense to the root parasite Heterodera glycines. Plant Signal Behav. 2015; 10(1):e977737.
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Transcriptome of Atoh7 retinal progenitor cells identifies new Atoh7-dependent regulatory genes for retinal ganglion cell formation. Dev Neurobiol. 2014 Nov; 74(11):1123-40.
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Ca2+-Calmodulin regulates SNARE assembly and spontaneous neurotransmitter release via v-ATPase subunit V0a1. J Cell Biol. 2014 Apr 14; 205(1):21-31.
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Syntaxin 31 functions in Glycine max resistance to the plant parasitic nematode Heterodera glycines. Plant Mol Biol. 2014 May; 85(1-2):107-21.
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ATP11B mediates platinum resistance in ovarian cancer. J Clin Invest. 2013 May; 123(5):2119-30.
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Syntaxin 6-mediated Golgi translocation plays an important role in nuclear functions of EGFR through microtubule-dependent trafficking. Oncogene. 2014 Feb 06; 33(6):756-70.