GTP-Binding Protein beta Subunits
"GTP-Binding Protein beta Subunits" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Heterotrimeric GTP-binding protein subunits that tightly associate with GTP-BINDING PROTEIN GAMMA SUBUNITS. A dimer of beta and gamma subunits is formed when the GTP-BINDING PROTEIN ALPHA SUBUNIT dissociates from the GTP-binding protein heterotrimeric complex. The beta-gamma dimer can play an important role in signal transduction by interacting with a variety of second messengers.
| Descriptor ID |
D044387
|
| MeSH Number(s) |
D12.644.360.360.520 D12.776.157.325.332.520 D12.776.476.375.520 D12.776.543.325.450
|
| Concept/Terms |
GTP-Binding Protein beta Subunits- GTP-Binding Protein beta Subunits
- GTP Binding Protein beta Subunits
- G-Protein beta Subunit
- G Protein beta Subunit
- Subunit, G-Protein beta
- beta Subunit, G-Protein
- GTP-Binding Protein beta Subunit
- GTP Binding Protein beta Subunit
|
Below are MeSH descriptors whose meaning is more general than "GTP-Binding Protein beta Subunits".
Below are MeSH descriptors whose meaning is more specific than "GTP-Binding Protein beta Subunits".
This graph shows the total number of publications written about "GTP-Binding Protein beta Subunits" by people in this website by year, and whether "GTP-Binding Protein beta Subunits" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2000 | 1 | 0 | 1 |
| 2003 | 1 | 0 | 1 |
| 2006 | 1 | 1 | 2 |
| 2007 | 2 | 0 | 2 |
| 2009 | 0 | 2 | 2 |
| 2010 | 2 | 0 | 2 |
| 2016 | 2 | 0 | 2 |
| 2017 | 0 | 2 | 2 |
To return to the timeline,
click here.
Below are the most recent publications written about "GTP-Binding Protein beta Subunits" by people in Profiles.
-
Ivermectin activates GIRK channels in a PIP2 -dependent, G?? -independent manner and an amino acid residue at the slide helix governs the activation. J Physiol. 2017 09 01; 595(17):5895-5912.
-
Lessons learned from additional research analyses of unsolved clinical exome cases. Genome Med. 2017 03 21; 9(1):26.
-
GNB5 Mutations Cause an Autosomal-Recessive Multisystem Syndrome with Sinus Bradycardia and Cognitive Disability. Am J Hum Genet. 2016 09 01; 99(3):704-710.
-
Germline De Novo Mutations in GNB1 Cause Severe Neurodevelopmental Disability, Hypotonia, and Seizures. Am J Hum Genet. 2016 05 05; 98(5):1001-1010.
-
The abundance and activation of mTORC1 regulators in skeletal muscle of neonatal pigs are modulated by insulin, amino acids, and age. J Appl Physiol (1985). 2010 Nov; 109(5):1448-54.
-
R9AP stabilizes RGS11-G beta5 and accelerates the early light response of ON-bipolar cells. Vis Neurosci. 2010 Mar; 27(1-2):9-17.
-
Distribution of RGS9-2 in neurons of the mouse striatum. J Neurochem. 2010 Feb; 112(3):651-61.
-
Two R7 regulator of G-protein signaling proteins shape retinal bipolar cell signaling. J Neurosci. 2009 Jun 17; 29(24):7753-65.
-
Gbeta5-RGS complexes co-localize with mGluR6 in retinal ON-bipolar cells. Eur J Neurosci. 2007 Nov; 26(10):2899-905.
-
The adaptor protein Lad associates with the G protein beta subunit and mediates chemokine-dependent T-cell migration. Blood. 2007 Jun 15; 109(12):5122-8.