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An Allosteric Shift in CD11c Affinity Activates a Proatherogenic State in Arrested Intermediate Monocytes.
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An Allosteric Shift in CD11c Affinity Activates a Proatherogenic State in Arrested Intermediate Monocytes.
An Allosteric Shift in CD11c Affinity Activates a Proatherogenic State in Arrested Intermediate Monocytes. J Immunol. 2020 11 15; 205(10):2806-2820.
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subject areas
Adult
Aged
Allosteric Regulation
Aorta
Case-Control Studies
CD11c Antigen
Cell Culture Techniques
Cell Line
Cell Membrane
Coronary Artery Disease
Coronary Vessels
Endothelial Cells
Endothelium, Vascular
Female
Humans
Integrin alpha4beta1
Lab-On-A-Chip Devices
Male
Microfluidic Analytical Techniques
Middle Aged
Monocytes
Non-ST Elevated Myocardial Infarction
Percutaneous Coronary Intervention
Recombinant Proteins
Transendothelial and Transepithelial Migration
Vascular Cell Adhesion Molecule-1
authors with profiles
HUAIZHU WU