"Endostatins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Angiostatic proteins that are formed from proteolytic cleavage of COLLAGEN TYPE XVIII.
| Descriptor ID |
D043169
|
| MeSH Number(s) |
D12.644.276.100.450.750 D12.776.467.100.450.750 D12.776.860.300.250.400.537.500 D23.529.100.450.750
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Endostatins".
Below are MeSH descriptors whose meaning is more specific than "Endostatins".
This graph shows the total number of publications written about "Endostatins" by people in this website by year, and whether "Endostatins" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2005 | 1 | 0 | 1 |
| 2007 | 0 | 1 | 1 |
| 2019 | 1 | 0 | 1 |
| 2025 | 1 | 0 | 1 |
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Below are the most recent publications written about "Endostatins" by people in Profiles.
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Counterbalancing angiogenic regulatory factors control the rate of cancer progression and survival in a stage-specific manner. Proc Natl Acad Sci U S A. 2011 Jun 14; 108(24):9939-44.
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Lack of collagen XVIII/endostatin exacerbates immune-mediated glomerulonephritis. J Am Soc Nephrol. 2010 Sep; 21(9):1445-55.
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Tumor stroma derived biomarkers in cancer. Cancer Metastasis Rev. 2009 Jun; 28(1-2):177-83.
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Cathepsin S controls angiogenesis and tumor growth via matrix-derived angiogenic factors. J Biol Chem. 2006 Mar 03; 281(9):6020-9.
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Function of endogenous inhibitors of angiogenesis as endothelium-specific tumor suppressors. Proc Natl Acad Sci U S A. 2005 Feb 22; 102(8):2934-9.
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Structural basis for the functions of endogenous angiogenesis inhibitors. Cold Spring Harb Symp Quant Biol. 2005; 70:399-410.
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The contribution of vascular basement membranes and extracellular matrix to the mechanics of tumor angiogenesis. APMIS. 2004 Jul-Aug; 112(7-8):450-62.
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Thrombospondin-1 associated with tumor microenvironment contributes to low-dose cyclophosphamide-mediated endothelial cell apoptosis and tumor growth suppression. Cancer Res. 2004 Mar 01; 64(5):1570-4.
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Cancer without disease. Nature. 2004 Feb 26; 427(6977):787.
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Quantitative analysis of biomarkers defines an optimal biological dose for recombinant human endostatin in primary human tumors. Clin Cancer Res. 2004 Jan 01; 10(1 Pt 1):33-42.