"Fructosediphosphates" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Diphosphoric acid esters of fructose. The fructose-1,6- diphosphate isomer is most prevalent. It is an important intermediate in the glycolysis process.
Descriptor ID |
D005635
|
MeSH Number(s) |
D09.894.417.313.300 D09.894.417.592.300
|
Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Fructosediphosphates".
Below are MeSH descriptors whose meaning is more specific than "Fructosediphosphates".
This graph shows the total number of publications written about "Fructosediphosphates" by people in this website by year, and whether "Fructosediphosphates" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
Year | Major Topic | Minor Topic | Total |
---|
2004 | 0 | 1 | 1 |
2007 | 1 | 0 | 1 |
2008 | 2 | 0 | 2 |
2010 | 1 | 0 | 1 |
2017 | 0 | 1 | 1 |
2018 | 0 | 1 | 1 |
To return to the timeline,
click here.
Below are the most recent publications written about "Fructosediphosphates" by people in Profiles.
-
EGFR-Phosphorylated Platelet Isoform of Phosphofructokinase 1 Promotes PI3K Activation. Mol Cell. 2018 04 19; 70(2):197-210.e7.
-
Benchmarking predictions of allostery in liver pyruvate kinase in CAGI4. Hum Mutat. 2017 09; 38(9):1123-1131.
-
Relevance of the ability of fructose 1,6-bis(phosphate) to sequester ferrous but not ferric ions. Carbohydr Res. 2011 Feb 15; 346(3):416-20.
-
Oral administration of fructose-1,6-diphosphate has anticonvulsant activity. Neurosci Lett. 2008 Dec 03; 446(2-3):75-7.
-
Pharmacokinetics of fructose-1,6-diphosphate after intraperitoneal and oral administration to adult rats. Pharmacol Res. 2008 Mar; 57(3):234-8.
-
Fructose-1,6-bisphosphate has anticonvulsant activity in models of acute seizures in adult rats. J Neurosci. 2007 Oct 31; 27(44):12007-11.
-
Energy failure in astrocytes increases the vulnerability of neurons to spreading depression. Eur J Neurosci. 2004 May; 19(9):2446-54.