"Response Elements" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Nucleotide sequences, usually upstream, which are recognized by specific regulatory transcription factors, thereby causing gene response to various regulatory agents. These elements may be found in both promoter and enhancer regions.
| Descriptor ID |
D020218
|
| MeSH Number(s) |
G02.111.570.080.689.330.700 G02.111.570.080.689.675.700 G05.360.080.689.330.700 G05.360.080.689.675.700 G05.360.340.024.340.137.750.249.765 G05.360.340.024.340.137.750.680.765
|
| Concept/Terms |
Response Elements- Response Elements
- Element, Response
- Elements, Response
- Response Element
|
Below are MeSH descriptors whose meaning is more general than "Response Elements".
Below are MeSH descriptors whose meaning is more specific than "Response Elements".
This graph shows the total number of publications written about "Response Elements" by people in this website by year, and whether "Response Elements" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1998 | 0 | 1 | 1 |
| 1999 | 3 | 2 | 5 |
| 2000 | 1 | 4 | 5 |
| 2001 | 0 | 5 | 5 |
| 2002 | 3 | 7 | 10 |
| 2003 | 1 | 4 | 5 |
| 2004 | 0 | 4 | 4 |
| 2005 | 1 | 16 | 17 |
| 2006 | 2 | 10 | 12 |
| 2007 | 1 | 8 | 9 |
| 2008 | 1 | 4 | 5 |
| 2009 | 2 | 5 | 7 |
| 2010 | 1 | 6 | 7 |
| 2011 | 3 | 5 | 8 |
| 2012 | 6 | 5 | 11 |
| 2013 | 2 | 4 | 6 |
| 2014 | 0 | 6 | 6 |
| 2015 | 0 | 2 | 2 |
| 2016 | 0 | 1 | 1 |
| 2017 | 0 | 1 | 1 |
| 2018 | 1 | 1 | 2 |
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Below are the most recent publications written about "Response Elements" by people in Profiles.
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Three classes of response elements for human PRC2 and MLL1/2-Trithorax complexes. Nucleic Acids Res. 2018 09 28; 46(17):8848-8864.
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Sox30 initiates transcription of haploid genes during late meiosis and spermiogenesis in mouse testes. Development. 2018 07 04; 145(13).
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Pharmacological targeting of MYC-regulated IRE1/XBP1 pathway suppresses MYC-driven breast cancer. J Clin Invest. 2018 04 02; 128(4):1283-1299.
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HN1L Promotes Triple-Negative Breast Cancer Stem Cells through LEPR-STAT3 Pathway. Stem Cell Reports. 2018 01 09; 10(1):212-227.
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A hypoxia response element in the Vegfa promoter is required for basal Vegfa expression in skin and for optimal granulation tissue formation during wound healing in mice. PLoS One. 2017; 12(7):e0180586.
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GGNBP2 acts as a tumor suppressor by inhibiting estrogen receptor a activity in breast cancer cells. Breast Cancer Res Treat. 2016 07; 158(2):263-76.
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Endocrine disrupting chemical, bisphenol-A, induces breast cancer associated gene HOXB9 expression in vitro and in vivo. Gene. 2016 Sep 30; 590(2):234-43.
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Use of Reporter Genes to Analyze Estrogen Response: The Transgenic Zebrafish Model. Methods Mol Biol. 2016; 1366:315-325.
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Chronic Hyperinsulinemia Causes Selective Insulin Resistance and Down-regulates Uncoupling Protein 3 (UCP3) through the Activation of Sterol Regulatory Element-binding Protein (SREBP)-1 Transcription Factor in the Mouse Heart. J Biol Chem. 2015 Dec 25; 290(52):30947-61.
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Elevated copper impairs hepatic nuclear receptor function in Wilson's disease. J Clin Invest. 2015 Sep; 125(9):3449-60.