Connection

SANJIV SUR to Animals

This is a "connection" page, showing publications SANJIV SUR has written about Animals.
Connection Strength

0.446
  1. Protocols to Measure Oxidative Stress and DNA Damage in Asthma. Methods Mol Biol. 2022; 2506:315-332.
    View in: PubMed
    Score: 0.035
  2. Excision release of 5?hydroxycytosine oxidatively induced DNA base lesions from the lung genome by cat dander extract challenge stimulates allergic airway inflammation. Clin Exp Allergy. 2018 12; 48(12):1676-1687.
    View in: PubMed
    Score: 0.028
  3. Attenuation of murine allergic airway inflammation with a CXCR1/CXCR2 chemokine receptor inhibitor. Clin Exp Allergy. 2019 01; 49(1):130-132.
    View in: PubMed
    Score: 0.028
  4. Role of epigenetics and DNA-damage in asthma. Curr Opin Allergy Clin Immunol. 2018 Feb; 18(1):32-37.
    View in: PubMed
    Score: 0.026
  5. Innate mechanism of pollen- and cat dander-induced oxidative stress and DNA damage in the airways. J Allergy Clin Immunol. 2017 11; 140(5):1436-1439.e5.
    View in: PubMed
    Score: 0.025
  6. Reply: Protease Plays a Role in Ragweed Pollen-Induced Neutrophil Recruitment and Epithelial Barrier Disruption. Am J Respir Cell Mol Biol. 2017 02; 56(2):272-273.
    View in: PubMed
    Score: 0.025
  7. Neutrophil recruitment by allergens contribute to allergic sensitization and allergic inflammation. Curr Opin Allergy Clin Immunol. 2016 Feb; 16(1):45-50.
    View in: PubMed
    Score: 0.023
  8. Facilitation of Allergic Sensitization and Allergic Airway Inflammation by Pollen-Induced Innate Neutrophil Recruitment. Am J Respir Cell Mol Biol. 2016 Jan; 54(1):81-90.
    View in: PubMed
    Score: 0.023
  9. Myeloid differentiation protein 2 facilitates pollen- and cat dander-induced innate and allergic airway inflammation. J Allergy Clin Immunol. 2016 05; 137(5):1506-1513.e2.
    View in: PubMed
    Score: 0.023
  10. Innate responses to pollen allergens. Curr Opin Allergy Clin Immunol. 2015 Feb; 15(1):79-88.
    View in: PubMed
    Score: 0.021
  11. Alternaria-induced release of IL-18 from damaged airway epithelial cells: an NF-?B dependent mechanism of Th2 differentiation? PLoS One. 2012; 7(2):e30280.
    View in: PubMed
    Score: 0.017
  12. Allergen challenge induces Ifng dependent GTPases in the lungs as part of a Th1 transcriptome response in a murine model of allergic asthma. PLoS One. 2009 Dec 21; 4(12):e8172.
    View in: PubMed
    Score: 0.015
  13. Inhibiting pollen reduced nicotinamide adenine dinucleotide phosphate oxidase-induced signal by intrapulmonary administration of antioxidants blocks allergic airway inflammation. J Allergy Clin Immunol. 2007 Mar; 119(3):646-53.
    View in: PubMed
    Score: 0.012
  14. ROS generated by pollen NADPH oxidase provide a signal that augments antigen-induced allergic airway inflammation. J Clin Invest. 2005 Aug; 115(8):2169-79.
    View in: PubMed
    Score: 0.011
  15. The DNA glycosylase NEIL2 is protective during SARS-CoV-2 infection. Nat Commun. 2023 Dec 09; 14(1):8169.
    View in: PubMed
    Score: 0.010
  16. In vivo role of p38 mitogen-activated protein kinase in mediating the anti-inflammatory effects of CpG oligodeoxynucleotide in murine asthma. J Immunol. 2002 Nov 15; 169(10):5955-61.
    View in: PubMed
    Score: 0.009
  17. Intrapulmonary administration of purified NEIL2 abrogates NF-?B-mediated inflammation. J Biol Chem. 2021 Jan-Jun; 296:100723.
    View in: PubMed
    Score: 0.008
  18. Mucosal bromodomain-containing protein 4 mediates aeroallergen-induced inflammation and remodeling. J Allergy Clin Immunol. 2019 04; 143(4):1380-1394.e9.
    View in: PubMed
    Score: 0.007
  19. Treating insect bite hypersensitivity in horses by using active vaccination against IL-5. J Allergy Clin Immunol. 2018 10; 142(4):1060-1061.
    View in: PubMed
    Score: 0.007
  20. Pollen-induced oxidative DNA damage response regulates miRNAs controlling allergic inflammation. Am J Physiol Lung Cell Mol Physiol. 2017 Dec 01; 313(6):L1058-L1068.
    View in: PubMed
    Score: 0.006
  21. Neil2-null Mice Accumulate Oxidized DNA Bases in the Transcriptionally Active Sequences of the Genome and Are Susceptible to Innate Inflammation. J Biol Chem. 2015 Oct 09; 290(41):24636-48.
    View in: PubMed
    Score: 0.006
  22. Whole transcriptome analysis reveals a role for OGG1-initiated DNA repair signaling in airway remodeling. Free Radic Biol Med. 2015 Dec; 89:20-33.
    View in: PubMed
    Score: 0.006
  23. 8-Oxoguanine DNA glycosylase-1-driven DNA base excision repair: role in asthma pathogenesis. Curr Opin Allergy Clin Immunol. 2015 Feb; 15(1):89-97.
    View in: PubMed
    Score: 0.005
  24. Whole transcriptome analysis reveals an 8-oxoguanine DNA glycosylase-1-driven DNA repair-dependent gene expression linked to essential biological processes. Free Radic Biol Med. 2015 Apr; 81:107-18.
    View in: PubMed
    Score: 0.005
  25. Innate inflammation induced by the 8-oxoguanine DNA glycosylase-1-KRAS-NF-?B pathway. J Immunol. 2014 Nov 01; 193(9):4643-53.
    View in: PubMed
    Score: 0.005
  26. The role of 8-oxoguanine DNA glycosylase-1 in inflammation. Int J Mol Sci. 2014 Sep 23; 15(9):16975-97.
    View in: PubMed
    Score: 0.005
  27. Effect of antigen sensitization and challenge on oscillatory mechanics of the lung and pulmonary inflammation in obese carboxypeptidase E-deficient mice. Am J Physiol Regul Integr Comp Physiol. 2014 Sep 15; 307(6):R621-33.
    View in: PubMed
    Score: 0.005
  28. 8-Oxoguanine DNA glycosylase-1-mediated DNA repair is associated with Rho GTPase activation and a-smooth muscle actin polymerization. Free Radic Biol Med. 2014 Aug; 73:430-8.
    View in: PubMed
    Score: 0.005
  29. Prevention of allergic rhinitis by aldose reductase inhibition in a murine model. Inflamm Allergy Drug Targets. 2013 Jun; 12(3):178-86.
    View in: PubMed
    Score: 0.005
  30. 8-Oxoguanine DNA glycosylase-1 links DNA repair to cellular signaling via the activation of the small GTPase Rac1. Free Radic Biol Med. 2013 Aug; 61:384-94.
    View in: PubMed
    Score: 0.005
  31. Down-regulation of 8-oxoguanine DNA glycosylase 1 expression in the airway epithelium ameliorates allergic lung inflammation. DNA Repair (Amst). 2013 Jan 01; 12(1):18-26.
    View in: PubMed
    Score: 0.005
  32. Biochemical identification of a hydroperoxide derivative of the free 8-oxo-7,8-dihydroguanine base. Free Radic Biol Med. 2012 Feb 15; 52(4):749-56.
    View in: PubMed
    Score: 0.004
  33. Mitochondrial dysfunction increases allergic airway inflammation. J Immunol. 2009 Oct 15; 183(8):5379-87.
    View in: PubMed
    Score: 0.004
  34. Aldose reductase inhibition suppresses the expression of Th2 cytokines and airway inflammation in ovalbumin-induced asthma in mice. J Immunol. 2009 Oct 01; 183(7):4723-32.
    View in: PubMed
    Score: 0.004
  35. Ragweed pollen-mediated IgE-independent release of biogenic amines from mast cells via induction of mitochondrial dysfunction. Mol Immunol. 2009 Aug; 46(13):2505-14.
    View in: PubMed
    Score: 0.004
  36. Divergent functions for airway epithelial matrix metalloproteinase 7 and retinoic acid in experimental asthma. Nat Immunol. 2009 May; 10(5):496-503.
    View in: PubMed
    Score: 0.004
  37. Iron-mediated dismutation of superoxide anion augments antigen-induced allergic inflammation: effect of lactoferrin. Postepy Hig Med Dosw (Online). 2007; 61:268-76.
    View in: PubMed
    Score: 0.003
  38. Subpollen particles: carriers of allergenic proteins and oxidases. J Allergy Clin Immunol. 2006 Oct; 118(4):844-50.
    View in: PubMed
    Score: 0.003
  39. Lactoferrin decreases pollen antigen-induced allergic airway inflammation in a murine model of asthma. Immunology. 2006 Oct; 119(2):159-66.
    View in: PubMed
    Score: 0.003
  40. Safe administration of a humanized murine antibody after anaphylaxis to a chimeric murine antibody. Transplantation. 2002 Dec 27; 74(12):1697-700.
    View in: PubMed
    Score: 0.002
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.