Central Amygdaloid Nucleus
"Central Amygdaloid Nucleus" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A nucleus located in the caudal half of the amygdala. It is a primary region involved in controlling responses and receives many intrinsic GABAergic amygdaloid connections.
Descriptor ID |
D066274
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MeSH Number(s) |
A08.186.211.464.090.750 A08.186.211.730.885.287.249.152.750
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Concept/Terms |
Central Amygdaloid Nucleus- Central Amygdaloid Nucleus
- Amygdaloid Nucleus, Central
- Nucleus, Central Amygdaloid
- Central Nucleus of the Amygdala
- Central Nucleus of Amygdala
- Amygdala Central Nucleus
- Central Amygdala Nucleus
- Amygdala Nucleus, Central
- Nucleus, Central Amygdala
- Central Amygdala
- Amygdala, Central
- Amygdalas, Central
- Central Amygdalas
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Below are MeSH descriptors whose meaning is more general than "Central Amygdaloid Nucleus".
Below are MeSH descriptors whose meaning is more specific than "Central Amygdaloid Nucleus".
This graph shows the total number of publications written about "Central Amygdaloid Nucleus" by people in this website by year, and whether "Central Amygdaloid Nucleus" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2015 | 1 | 0 | 1 |
2022 | 1 | 0 | 1 |
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Below are the most recent publications written about "Central Amygdaloid Nucleus" by people in Profiles.
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CGRP-dependent sensitization of PKC-d positive neurons in central amygdala mediates chronic migraine. J Headache Pain. 2022 Dec 12; 23(1):157.
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IL-10 normalizes aberrant amygdala GABA transmission and reverses anxiety-like behavior and dependence-induced escalation of alcohol intake. Prog Neurobiol. 2021 04; 199:101952.
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SIRT1 Decreases Emotional Pain Vulnerability with Associated CaMKIIa Deacetylation in Central Amygdala. J Neurosci. 2020 03 11; 40(11):2332-2342.
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GluA1 in central amygdala increases pain but inhibits opioid withdrawal-induced aversion. Mol Pain. 2020 Jan-Dec; 16:1744806920911543.
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GluA1 in Central Amygdala Promotes Opioid Use and Reverses Inhibitory Effect of Pain. Neuroscience. 2020 02 01; 426:141-153.
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A Central Amygdala Input to the Parafascicular Nucleus Controls Comorbid Pain in Depression. Cell Rep. 2019 12 17; 29(12):3847-3858.e5.
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Protein Kinase C Epsilon Activity in the Nucleus Accumbens and Central Nucleus of the Amygdala Mediates Binge Alcohol Consumption. Biol Psychiatry. 2016 Mar 15; 79(6):443-51.
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Persistent pain facilitates response to morphine reward by downregulation of central amygdala GABAergic function. Neuropsychopharmacology. 2014 Aug; 39(9):2263-71.