Connection

Co-Authors

This is a "connection" page, showing publications co-authored by XANDER WEHRENS and SATADRU LAHIRI.
Connection Strength

6.338
  1. Altered myocardial lipid regulation in junctophilin-2-associated familial cardiomyopathies. Life Sci Alliance. 2024 May; 7(5).
    View in: PubMed
    Score: 0.991
  2. Cellular regeneration as a potential strategy to treat cardiac conduction disorders. J Clin Invest. 2021 10 01; 131(19).
    View in: PubMed
    Score: 0.838
  3. Correction to: mechanisms underlying pathological Ca2+ handling in diseases of the heart. Pflugers Arch. 2021 Mar; 473(3):349.
    View in: PubMed
    Score: 0.805
  4. Mechanisms underlying pathological Ca2+ handling in diseases of the heart. Pflugers Arch. 2021 03; 473(3):331-347.
    View in: PubMed
    Score: 0.796
  5. Nuclear localization of a novel calpain-2 mediated junctophilin-2 C-terminal cleavage peptide promotes cardiomyocyte remodeling. Basic Res Cardiol. 2020 06 26; 115(4):49.
    View in: PubMed
    Score: 0.768
  6. Atrial Proteomic Profiling Reveals a Switch Towards Profibrotic Gene Expression Program in CREM-Ib?C-X Mice with Persistent Atrial Fibrillation. bioRxiv. 2024 Jan 12.
    View in: PubMed
    Score: 0.245
  7. Long-term efficacy and safety of cardiac genome editing for catecholaminergic polymorphic ventricular tachycardia. J Cardiovasc Aging. 2024 Jan; 4(1).
    View in: PubMed
    Score: 0.245
  8. Characterization of atrial and ventricular remodeling in an improved minimally invasive mouse model of transverse aortic constriction. J Cardiovasc Aging. 2023 Jul; 3(3).
    View in: PubMed
    Score: 0.237
  9. Targeting calcium-mediated inter-organellar crosstalk in cardiac diseases. Expert Opin Ther Targets. 2022 04; 26(4):303-317.
    View in: PubMed
    Score: 0.218
  10. Atrial-Specific LKB1 Knockdown Represents a Novel Mouse Model of Atrial Cardiomyopathy With Spontaneous Atrial Fibrillation. Circulation. 2021 09 14; 144(11):909-912.
    View in: PubMed
    Score: 0.209
  11. Phosphorylation-Dependent Interactome of Ryanodine Receptor Type 2 in the Heart. Proteomes. 2021 Jun 07; 9(2).
    View in: PubMed
    Score: 0.205
  12. Rearrangement of the Protein Phosphatase 1 Interactome During Heart Failure Progression. Circulation. 2018 10 09; 138(15):1569-1581.
    View in: PubMed
    Score: 0.170
  13. In Vivo Ryr2 Editing Corrects Catecholaminergic Polymorphic Ventricular Tachycardia. Circ Res. 2018 Sep 28; 123(8):953-963.
    View in: PubMed
    Score: 0.170
  14. AKAP12 Upregulation Associates With PDE8A to Accelerate Cardiac Dysfunction. Circ Res. 2024 Apr 12; 134(8):1006-1022.
    View in: PubMed
    Score: 0.062
  15. Atrial proteomic profiling reveals a switch towards profibrotic gene expression program in CREM-Ib?C-X mice with persistent atrial fibrillation. J Mol Cell Cardiol. 2024 Mar 19; 190:1-12.
    View in: PubMed
    Score: 0.062
  16. Chronic kidney disease promotes atrial fibrillation via inflammasome pathway activation. J Clin Invest. 2023 10 02; 133(19).
    View in: PubMed
    Score: 0.060
  17. Junctophilin-2 Regulates Mitochondrial Metabolism. bioRxiv. 2023 Feb 08.
    View in: PubMed
    Score: 0.058
  18. Genetic inhibition of nuclear factor of activated T-cell c2 prevents atrial fibrillation in CREM transgenic mice. Cardiovasc Res. 2022 10 21; 118(13):2805-2818.
    View in: PubMed
    Score: 0.056
  19. Paracrine signalling by cardiac calcitonin controls atrial fibrogenesis and arrhythmia. Nature. 2020 11; 587(7834):460-465.
    View in: PubMed
    Score: 0.049
  20. Loss of SPEG Inhibitory Phosphorylation of Ryanodine Receptor Type-2 Promotes Atrial Fibrillation. Circulation. 2020 09 22; 142(12):1159-1172.
    View in: PubMed
    Score: 0.048
  21. Loss of Protein Phosphatase 1 Regulatory Subunit PPP1R3A Promotes Atrial Fibrillation. Circulation. 2019 08 20; 140(8):681-693.
    View in: PubMed
    Score: 0.045
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.